Guido C. Faas - Publications

Affiliations: 
University of California, Los Angeles, Los Angeles, CA 
Area:
calcium binding proteins, calcium kinetics, ion signalling

17 high-probability publications. We are testing a new system for linking publications to authors. You can help! If you notice any innacuracies, please sign in and mark papers as correct or incorrect matches. If you identify any major omissions or other inaccuracies in the publication list, please let us know.

Year Citation  Score
2016 Ferando I, Faas GC, Mody I. Diminished KCC2 confounds synapse specificity of LTP during senescence. Nature Neuroscience. PMID 27500406 DOI: 10.1038/nn.4357  1
2015 Chen L, Faas GC, Ferando I, Mody I. Novel insights into the behavioral analysis of mice subjected to the forced-swim test. Translational Psychiatry. 5: e551. PMID 25871976 DOI: 10.1038/tp.2015.44  1
2014 Faas GC, Mody I. Measuring the Ca²⁺-binding kinetics of proteins. Cold Spring Harbor Protocols. 2014: 691-3. PMID 24987145 DOI: 10.1101/pdb.top066258  1
2014 Faas GC, Mody I. Collecting data to determine the Ca²⁺-binding properties of DM-nitrophen and proteins. Cold Spring Harbor Protocols. 2014: 768-73. PMID 24987136 DOI: 10.1101/pdb.prot073296  1
2014 Faas GC, Mody I. Measuring the rate constants of Ca²⁺ indicators. Cold Spring Harbor Protocols. 2014: 763-7. PMID 24987135 DOI: 10.1101/pdb.prot073288  1
2014 Faas GC, Mody I. Measuring the steady-state properties of Ca²⁺ indicators with a set of calibrated [Ca²⁺] solutions. Cold Spring Harbor Protocols. 2014: 758-62. PMID 24987134 DOI: 10.1101/pdb.prot073270  1
2014 Tong X, Ao Y, Faas GC, Nwaobi SE, Xu J, Haustein MD, Anderson MA, Mody I, Olsen ML, Sofroniew MV, Khakh BS. Astrocyte Kir4.1 ion channel deficits contribute to neuronal dysfunction in Huntington's disease model mice. Nature Neuroscience. 17: 694-703. PMID 24686787 DOI: 10.1038/nn.3691  1
2014 Jones RT, Faas GC, Mody I. Intracellular bicarbonate regulates action potential generation via KCNQ channel modulation. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 34: 4409-17. PMID 24647960 DOI: 10.1523/JNEUROSCI.3836-13.2014  1
2012 Faas GC, Mody I. Measuring the kinetics of calcium binding proteins with flash photolysis. Biochimica Et Biophysica Acta. 1820: 1195-204. PMID 22001612 DOI: 10.1016/j.bbagen.2011.09.012  1
2011 Faas GC, Raghavachari S, Lisman JE, Mody I. Calmodulin as a direct detector of Ca2+ signals. Nature Neuroscience. 14: 301-4. PMID 21258328 DOI: 10.1038/nn.2746  1
2007 Faas GC, Schwaller B, Vergara JL, Mody I. Resolving the fast kinetics of cooperative binding: Ca2+ buffering by calretinin. Plos Biology. 5: e311. PMID 18044987 DOI: 10.1371/journal.pbio.0050311  1
2005 Faas GC, Karacs K, Vergara JL, Mody I. Kinetic properties of DM-nitrophen binding to calcium and magnesium. Biophysical Journal. 88: 4421-33. PMID 15778435 DOI: 10.1529/biophysj.104.057745  1
2003 Levy M, Faas GC, Saggau P, Craigen WJ, Sweatt JD. Mitochondrial regulation of synaptic plasticity in the hippocampus. The Journal of Biological Chemistry. 278: 17727-34. PMID 12604600 DOI: 10.1074/jbc.M212878200  1
2002 Faas GC, Adwanikar H, Gereau RW, Saggau P. Modulation of presynaptic calcium transients by metabotropic glutamate receptor activation: a differential role in acute depression of synaptic transmission and long-term depression. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 22: 6885-90. PMID 12177186  1
1998 Vreugdenhil M, Faas GC, Wadman WJ. Sodium currents in isolated rat CA1 neurons after kindling epileptogenesis. Neuroscience. 86: 99-107. PMID 9692746 DOI: 10.1016/S0306-4522(98)00041-4  1
1996 Faas GC, Vreugdenhil M, Wadman WJ. Calcium currents in pyramidal CA1 neurons in vitro after kindling epileptogenesis in the hippocampus of the rat. Neuroscience. 75: 57-67. PMID 8923523 DOI: 10.1016/0306-4522(96)00254-0  1
1993 Somjen GG, Faas GC, Vreugdenhil M, Wadman WJ. Channel shutdown: a response of hippocampal neurons to adverse environments. Brain Research. 632: 180-94. PMID 7511975 DOI: 10.1016/0006-8993(93)91153-J  1
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