Michael S. Levine

Affiliations: 
University of California, Los Angeles, Los Angeles, CA 
Area:
huntington's disease, neurophysiology
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"Michael Levine"
Mean distance: 14.55 (cluster 6)
 

Parents

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Alan Garen grad student 1981 (FlyTree)
Walter Gehring post-doc 1982-1983
Nathaniel A. Buchwald research scientist UCLA

Children

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Mary Kay Lobo research assistant Brain Research Institute, UCLA
Viorela Pop research assistant 2001-2002 UCLA
Benjamin Lacar research assistant 2003-2004 Brain Research Institute, UCLA
Takahiro Soda research assistant 2002-2005 Brain Research Institute, UCLA
Besim Omer Uzgil research assistant 2002-2005 UCLA
Max Kleiman-Weiner research assistant 2004-2006 Brain Research Institute, UCLA
Bianca Marcolino research assistant 2005-2006 Brain Research Institute, UCLA
Joseph P. Antonios research assistant 2007-2011 UCLA
Joshua L. Plotkin grad student UCLA
Jorge Flores-Hernandez grad student 1998-2001 UCLA
Emily L. Jocoy grad student 2009 UCLA
Jason S. Hauptman grad student 2011 UCLA
Stephen M. Siviy post-doc UCLA
Howard Casey Cromwell post-doc 1993-1996 Brain Research Institute, UCLA
Gloria Janine Klapstein post-doc 1998-2004 UCLA
Damian M. Cummings post-doc 2004-2009 Brain Research Institute, UCLA
David A. Hendrix post-doc 2007-2010 UC Berkeley (Physics Tree)
Sandra M. Holley post-doc 2009-2013 UCLA
Veronique Marie Andre research scientist 2003- UCLA
BETA: Related publications

Publications

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Barry J, Sarafian TA, Watson JB, et al. (2020) Mechanisms underlying the enhancement of γ-aminobutyric acid responses in the external globus pallidus of R6/2 Huntington's disease model mice. Journal of Neuroscience Research
Levinson S, Tran CH, Barry J, et al. (2020) Paroxysmal Discharges in Tissue Slices From Pediatric Epilepsy Surgery Patients: Critical Role of GABA Receptors in the Generation of Ictal Activity. Frontiers in Cellular Neuroscience. 14: 54
Donzis EJ, Estrada-Sánchez AM, Indersmitten T, et al. (2019) Cortical Network Dynamics Is Altered in Mouse Models of Huntington's Disease. Cerebral Cortex (New York, N.Y. : 1991)
Lee K, Bakhurin KI, Claar LD, et al. (2019) Gain Modulation by Corticostriatal and Thalamostriatal Input Signals during Reward-Conditioned Behavior. Cell Reports. 29: 2438-2449.e4
Holley SM, Galvan L, Kamdjou T, et al. (2019) Major Contribution of Somatostatin-Expressing Interneurons and Cannabinoid Receptors to Increased GABA Synaptic Activity in the Striatum of Huntington's Disease Mice. Frontiers in Synaptic Neuroscience. 11: 14
Cepeda C, Levinson S, Yazon VW, et al. (2018) Cellular antiseizure mechanisms of everolimus in pediatric tuberous sclerosis complex, cortical dysplasia, and non-mTOR-mediated etiologies. Epilepsia Open. 3: 180-190
Holley SM, Galvan L, Kamdjou T, et al. (2018) Striatal GABAergic Interneuron Dysfunction in the Q175 Mouse Model of Huntington's Disease. The European Journal of Neuroscience
Wood TE, Barry J, Yang Z, et al. (2018) Mutant huntingtin reduction in astrocytes slows disease progression in the bachd conditional huntington's disease mouse model. Human Molecular Genetics
Estrada-Sánchez AM, Castro D, Portillo-Ortiz K, et al. (2018) Complete but not partial inhibition of glutamate transporters exacerbates cortical excitability in the R6/2 mouse model of Huntington's disease. Cns Neuroscience & Therapeutics
Shin BC, Cepeda C, María Estrada-Sánchez A, et al. (2018) Neural Deletion of Glucose Transporter Isoform 3 Creates Distinct Postnatal and Adult Neurobehavioral Phenotypes. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience
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