Damian M. Cummings
Affiliations: | 1997-2000 | Physiology | University College London |
2000-2004 | Biomedical Sciences | The Open University, Kents Hill, England, United Kingdom | |
2004-2009 | Mental Retardation Research Center | University of California, Los Angeles, Los Angeles, CA | |
2009- | Neuroscience, Physiology & Pharmacology | University College London, London, United Kingdom |
Area:
Electrophysiology, Dementia, Alzheimer's disease, Huntington's disease, Long-term potentiation, Synaptic plasticity,Google:
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Parents
Sign in to add mentorMicheal G. Stewart | grad student | The Open University | ||
Lynn J. Bindman | grad student | 1999-2000 | UCL | |
(Undergraduate and summer project student) | ||||
Kerry PSJ Murphy | grad student | 2000-2004 | Open University, Milton Keynes, England | |
(PhD student) | ||||
Michael S. Levine | post-doc | 2004-2009 | Brain Research Institute, UCLA | |
(Postdoc) | ||||
Frances Edwards | research scientist | 2009- | UCL (London) |
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Publications
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Desai S, Camporesi E, Brinkmalm G, et al. (2024) Age- and amyloid-β-dependent initiation of neurofibrillary tau tangles: an improved mouse model of Alzheimer's disease without mutations in . Biorxiv : the Preprint Server For Biology |
Wood JI, Dulewicz M, Ge J, et al. (2024) Isotope Encoded chemical Imaging Identifies Amyloid Plaque Age Dependent Structural Maturation, Synaptic Loss, and Increased Toxicity. Biorxiv : the Preprint Server For Biology |
Wood JI, Wong E, Joghee R, et al. (2022) Plaque contact and unimpaired Trem2 is required for the microglial response to amyloid pathology. Cell Reports. 41: 111686 |
Benitez DP, Jiang S, Wood J, et al. (2021) Knock-in models related to Alzheimer's disease: synaptic transmission, plaques and the role of microglia. Molecular Neurodegeneration. 16: 47 |
Liu W, Taso O, Wang R, et al. (2020) Trem2 promotes anti-inflammatory responses in microglia and is suppressed under pro-inflammatory conditions. Human Molecular Genetics |
Salih DA, Bayram S, Guelfi S, et al. (2019) Genetic variability in response to amyloid beta deposition influences Alzheimer's disease risk. Brain Communications. 1: fcz022 |
Vitanova KS, Stringer KM, Benitez DP, et al. (2019) Dementia associated with disorders of the basal ganglia. Journal of Neuroscience Research |
Cepeda C, Oikonomou KD, Cummings D, et al. (2019) Developmental origins of cortical hyperexcitability in Huntington's disease: Review and new observations. Journal of Neuroscience Research |
Joel Z, Izquierdo P, Salih DA, et al. (2019) Improving Mouse Models for Dementia. Are All the Effects in Tau Mouse Models Due to Overexpression? Cold Spring Harbor Symposia On Quantitative Biology |
Medawar E, Benway TA, Liu W, et al. (2018) Effects of rising amyloidβ levels on hippocampal synaptic transmission, microglial response and cognition in APP/PSEN1 transgenic mice. Ebiomedicine |