Stephanie S. Minami, Ph.D.

Affiliations: 
Neuroscience Georgetown University, Washington, DC 
Area:
Molecular mechanisms of Alzheimer's disease
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"Stephanie Minami"
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G. William Rebeck grad student 2010 Georgetown
 (The role of Fyn in the pathogenic processes of Alzheimer's disease.)
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Publications

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Min SW, Chen X, Tracy TE, et al. (2015) Critical role of acetylation in tau-mediated neurodegeneration and cognitive deficits. Nature Medicine
Minami SS, Min SW, Krabbe G, et al. (2014) Progranulin protects against amyloid β deposition and toxicity in Alzheimer's disease mouse models. Nature Medicine. 20: 1157-64
Minami SS, Clifford TG, Hoe HS, et al. (2012) Fyn knock-down increases Aβ, decreases phospho-tau, and worsens spatial learning in 3×Tg-AD mice. Neurobiology of Aging. 33: 825.e15-24
Babus LW, Little EM, Keenoy KE, et al. (2011) Decreased dendritic spine density and abnormal spine morphology in Fyn knockout mice. Brain Research. 1415: 96-102
Minami SS, Hoe HS, Rebeck GW. (2011) Fyn kinase regulates the association between amyloid precursor protein and Dab1 by promoting their localization to detergent-resistant membranes Journal of Neurochemistry. 118: 879-890
Minami SS, Sidahmed E, Aid S, et al. (2010) Therapeutic versus neuroinflammatory effects of passive immunization is dependent on Aβ/amyloid burden in a transgenic mouse model of Alzheimer's disease. Journal of Neuroinflammation. 7: 57
Minami SS, Cordova A, Cirrito JR, et al. (2010) ApoE mimetic peptide decreases Abeta production in vitro and in vivo. Molecular Neurodegeneration. 5: 16
Minami SS, Sung YM, Dumanis SB, et al. (2010) The cytoplasmic adaptor protein X11alpha and extracellular matrix protein Reelin regulate ApoE receptor 2 trafficking and cell movement. Faseb Journal : Official Publication of the Federation of American Societies For Experimental Biology. 24: 58-69
Minami SS, Hoe HS, Burns MP, et al. (2009) P4-216: Fyn kinase regulates phosphorylation and localization of APP and Dab1 to lipid rafts Alzheimer's & Dementia. 5: P494-P494
Hirata-Fukae C, Li HF, Hoe HS, et al. (2008) Females exhibit more extensive amyloid, but not tau, pathology in an Alzheimer transgenic model. Brain Research. 1216: 92-103
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