Ricardo E. Dolmetsch

Stanford University, Palo Alto, CA 
"Ricardo Dolmetsch"
Mean distance: 13.71 (cluster 11)
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Panagiotakos G, Haveles C, Arjun A, et al. (2019) Aberrant calcium channel splicing drives defects in cortical differentiation in Timothy Syndrome. Elife. 8
Sun Y, Paşca SP, Portmann T, et al. (2016) A deleterious Nav1.1 mutation selectively impairs telencephalic inhibitory neurons derived from Dravet Syndrome patients. Elife. 5
Tsuruta F, Dolmetsch RE. (2015) PIKfyve mediates the motility of late endosomes and lysosomes in neuronal dendrites. Neuroscience Letters. 605: 18-23
Rana A, Yen M, Sadaghiani AM, et al. (2015) Alternative splicing converts STIM2 from an activator to an inhibitor of store-operated calcium channels. The Journal of Cell Biology. 209: 653-70
Byers B, Lee HJ, Liu J, et al. (2015) Direct in vivo assessment of human stem cell graft-host neural circuits. Neuroimage. 114: 328-37
Yang M, Mahrt EJ, Lewis F, et al. (2015) 16p11.2 Deletion Syndrome Mice Display Sensory and Ultrasonic Vocalization Deficits During Social Interactions. Autism Research : Official Journal of the International Society For Autism Research
Tian Y, Voineagu I, Pa?ca SP, et al. (2014) Alteration in basal and depolarization induced transcriptional network in iPSC derived neurons from Timothy syndrome. Genome Medicine. 6: 75
Sadaghiani AM, Lee SM, Odegaard JI, et al. (2014) Identification of Orai1 channel inhibitors by using minimal functional domains to screen small molecule microarrays. Chemistry & Biology. 21: 1278-92
PaÅŸca SP, Panagiotakos G, Dolmetsch RE. (2014) Generating human neurons in vitro and using them to understand neuropsychiatric disease. Annual Review of Neuroscience. 37: 479-501
Portmann T, Yang M, Mao R, et al. (2014) Behavioral abnormalities and circuit defects in the basal ganglia of a mouse model of 16p11.2 deletion syndrome. Cell Reports. 7: 1077-92
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