Elizabeth S. Yeh, Ph.D.

Affiliations: 
2004 Duke University, Durham, NC 
Area:
cell signaling cascades that regulate cell proliferation, differentiation or function
Google:
"Elizabeth Yeh"
Mean distance: 19.23 (cluster 11)
 
SNBCP

Parents

Sign in to add mentor
Anthony R. Means grad student 2004 Duke
 (Identification c-Myc and cyclin E as novel Pin1 substrates and the role of Pin1 in cellular transformation.)
BETA: Related publications

Publications

You can help our author matching system! If you notice any publications incorrectly attributed to this author, please sign in and mark matches as correct or incorrect.

Yeh ES, Belka GK, Vernon AE, et al. (2013) Hunk negatively regulates c-myc to promote Akt-mediated cell survival and mammary tumorigenesis induced by loss of Pten. Proceedings of the National Academy of Sciences of the United States of America. 110: 6103-8
Alvarez JV, Yeh ES, Feng Y, et al. (2012) Oncogene addiction: Mouse models and clinical relevance for molecularly targeted therapies Genetically Engineered Mice For Cancer Research: Design, Analysis, Pathways, Validation and Pre-Clinical Testing. 527-547
Yeh ES, Yang TW, Jung JJ, et al. (2011) Hunk is required for HER2/neu-induced mammary tumorigenesis. The Journal of Clinical Investigation. 121: 866-79
Yeh ES, Means AR. (2007) PIN1, the cell cycle and cancer. Nature Reviews. Cancer. 7: 381-8
van Drogen F, Sangfelt O, Malyukova A, et al. (2006) Ubiquitylation of cyclin E requires the sequential function of SCF complexes containing distinct hCdc4 isoforms. Molecular Cell. 23: 37-48
Yeh ES, Lew BO, Means AR. (2006) The loss of PIN1 deregulates cyclin E and sensitizes mouse embryo fibroblasts to genomic instability. The Journal of Biological Chemistry. 281: 241-51
Yeh E, Cunningham M, Arnold H, et al. (2004) A signalling pathway controlling c-Myc degradation that impacts oncogenic transformation of human cells. Nature Cell Biology. 6: 308-18
Joseph JD, Yeh ES, Swenson KI, et al. (2003) The peptidyl-prolyl isomerase Pin1. Progress in Cell Cycle Research. 5: 477-87
See more...