Taihao Jin, Ph.D.

2003 Icahn School of Medicine at Mount Sinai, New York, NY, United States 
ionic channels, physiology
"Taihao Jin"
Mean distance: 17.49 (cluster 32)


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Diomedes E. Logothetis grad student 2003 Mount Sinai School of Medicine
 (The beta gamma subunits of G proteins gate a potassium channel by pivoted bending of a transmembrane segment.)
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Li D, Jin T, Gazgalis D, et al. (2019) On the mechanism of the GIRK2 channel gating by phosphatidylinositol bisphosphate (PIP2), sodium, and the Gβγ dimer. The Journal of Biological Chemistry
Sun Y, Dong Z, Jin T, et al. (2013) Imaging-based chemical screening reveals activity-dependent neural differentiation of pluripotent stem cells. Elife. 2: e00508
Yang H, Kim A, David T, et al. (2012) TMEM16F forms a Ca2+-activated cation channel required for lipid scrambling in platelets during blood coagulation. Cell. 151: 111-22
Yang H, Jin T, Cheng T, et al. (2011) Scan: A Novel Small-Conductance Ca2+-Activated Non-Selective Cation Channel Encoded by TMEM16F Biophysical Journal. 100: 259a
Styer AM, Mirshahi UL, Wang C, et al. (2010) G protein {beta}{gamma} gating confers volatile anesthetic inhibition to Kir3 channels. The Journal of Biological Chemistry. 285: 41290-9
Jin T, Sui JL, Rosenhouse-Dantsker A, et al. (2008) Stoichiometry of Kir channels with phosphatidylinositol bisphosphate. Channels (Austin, Tex.). 2: 19-33
deHart GW, Jin T, McCloskey DE, et al. (2008) The alpha9beta1 integrin enhances cell migration by polyamine-mediated modulation of an inward-rectifier potassium channel. Proceedings of the National Academy of Sciences of the United States of America. 105: 7188-93
Logothetis DE, Jin T, Lupyan D, et al. (2007) Phosphoinositide-mediated gating of inwardly rectifying K(+) channels. PflüGers Archiv : European Journal of Physiology. 455: 83-95
Mirshahi T, Jin T, Logothetis DE. (2003) G beta gamma and KACh: old story, new insights. Science's Stke : Signal Transduction Knowledge Environment. 2003: PE32
Zhang H, Craciun LC, Mirshahi T, et al. (2003) PIP(2) activates KCNQ channels, and its hydrolysis underlies receptor-mediated inhibition of M currents. Neuron. 37: 963-75
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