John P. Adelman
Affiliations: | Vollum Institute | Oregon Health and Science University, Portland, OR |
Area:
synaptic physiologyWebsite:
http://www.ohsu.edu/xd/research/centers-institutes/vollum/faculty/adelmanlab.cfmGoogle:
"John Adelman"Mean distance: 14.18 (cluster 6)
Children
Sign in to add trainee| Thanos Tzounopoulos | grad student | OHSU | |
| Michael D. Varnum | grad student | OHSU & Vollum | |
| Andrew Bruening-Wright | grad student | 2003 | OHSU |
| Chao Sun | post-doc | UC Davis | |
| Stephen J. Tucker | post-doc | OHSU |
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Publications
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| Raghuram V, Weber S, Raber J, et al. (2017) Assessment of mutations in KCNN2 and ZNF135 to patient neurological symptoms. Neuroreport |
| Grasselli G, He Q, Wan V, et al. (2016) Activity-Dependent Plasticity of Spike Pauses in Cerebellar Purkinje Cells. Cell Reports |
| Yap FC, Weber DS, Taylor MS, et al. (2016) Endothelial SK3 channel-associated Ca2+ microdomains modulate blood pressure. American Journal of Physiology. Heart and Circulatory Physiology. ajpheart.00787.2015 |
| Kim G, Luján R, Schwenk J, et al. (2016) Membrane palmitoylated protein 2 is a synaptic scaffold protein required for synaptic SK2-containing channel function. Elife. 5 |
| Wang K, Mateos-Aparicio P, Hönigsperger C, et al. (2016) IK1 channels do not contribute to the slow afterhyperpolarization in pyramidal neurons. Elife. 5 |
| Wang K, Kelley MH, Wu WW, et al. (2015) Apamin Boosting of Synaptic Potentials in CaV2.3 R-Type Ca2+ Channel Null Mice. Plos One. 10: e0139332 |
| Adelman JP. (2015) SK channels and calmodulin. Channels (Austin, Tex.). 1-6 |
| Ballesteros-Merino C, Martínez-Hernández J, Aguado C, et al. (2014) Localization of SK2 channels relative to excitatory synaptic sites in the mouse developing Purkinje cells. Frontiers in Neuroanatomy. 8: 154 |
| GarcÃa-Negredo G, Soto D, Llorente J, et al. (2014) Coassembly and coupling of SK2 channels and mGlu5 receptors. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 34: 14793-802 |
| Orfila JE, Shimizu K, Garske AK, et al. (2014) Increasing small conductance Ca2+-activated potassium channel activity reverses ischemia-induced impairment of long-term potentiation. The European Journal of Neuroscience. 40: 3179-88 |