George W. Huntley

Icahn School of Medicine at Mount Sinai, New York, NY, United States 
Synapse development/plasticity
"George Huntley"
Mean distance: 13.4 (cluster 6)
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Hussein A, Tielemans A, Baxter MG, et al. (2022) Cognitive deficits and altered cholinergic innervation in young adult male mice carrying a Parkinson's disease Lrrk2 knockin mutation. Experimental Neurology. 355: 114145
de la Fuente Revenga M, Zhu B, Guevara CA, et al. (2021) Prolonged epigenomic and synaptic plasticity alterations following single exposure to a psychedelic in mice. Cell Reports. 37: 109836
Hussein A, Guevara CA, Del Valle P, et al. (2021) Non-Motor Symptoms of Parkinson's Disease: The Neurobiology of Early Psychiatric and Cognitive Dysfunction. The Neuroscientist : a Review Journal Bringing Neurobiology, Neurology and Psychiatry. 10738584211011979
Guevara CA, Matikainen-Ankney BA, Kezunovic N, et al. (2020) LRRK2 mutation alters behavioral, synaptic and non-synaptic adaptations to acute social stress. Journal of Neurophysiology
Flanigan ME, Aleyasin H, Li L, et al. (2020) Orexin signaling in GABAergic lateral habenula neurons modulates aggressive behavior in male mice. Nature Neuroscience
Huntley GW, Benson DL. (2020) Origins of Parkinson's Disease in Brain Development: Insights From Early and Persistent Effects of LRRK2-G2019S on Striatal Circuits. Frontiers in Neuroscience. 14: 265
Benson DL, Huntley GW. (2019) Are We Listening to Everything the PARK Genes are Telling Us? The Journal of Comparative Neurology
Benson DL, Matikainen-Ankney BA, Hussein A, et al. (2018) Functional and behavioral consequences of Parkinson's disease-associated G2019S mutation. Biochemical Society Transactions
Matikainen-Ankney BA, Kezunovic N, Menard C, et al. (2018) Parkinson's Disease-linked LRRK2-G2019S mutation alters synaptic plasticity and promotes resilience to chronic social stress in young adulthood. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience
Ibi D, de la Fuente Revenga M, Kezunovic N, et al. (2017) Antipsychotic-induced Hdac2 transcription via NF-κB leads to synaptic and cognitive side effects. Nature Neuroscience
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