BETA: Related publications

Publications

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Polster A, Dittmer PJ, Perni S, et al. (2018) Stac Proteins Suppress Ca-Dependent Inactivation of Neuronal L-type Ca Channels. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience
Polster A, Nelson BR, Papadopoulos S, et al. (2018) Stac proteins associate with the critical domain for excitation-contraction coupling in the II-III loop of Ca1.1. The Journal of General Physiology
Polster A, Perni S, Filipova D, et al. (2017) Junctional trafficking and restoration of retrograde signaling by the cytoplasmic RyR1 domain. The Journal of General Physiology
Perni S, Lavorato M, Beam KG. (2017) De novo reconstitution reveals the proteins required for skeletal muscle voltage-induced Ca2+ release. Proceedings of the National Academy of Sciences of the United States of America
Polster A, Nelson BR, Olson EN, et al. (2016) Stac3 has a direct role in skeletal muscle-type excitation-contraction coupling that is disrupted by a myopathy-causing mutation. Proceedings of the National Academy of Sciences of the United States of America
Bannister RA, Sheridan DC, Beam KG. (2016) Distinct Components of Retrograde CaV1.1-RyR1 Coupling Revealed by a Lethal Mutation in RyR1. Biophysical Journal. 110: 912-21
Ohrtman JD, Romberg CF, Moua O, et al. (2015) Apparent lack of physical or functional interaction between CaV1.1 and its distal C terminus. The Journal of General Physiology. 145: 303-14
Polster A, Perni S, Bichraoui H, et al. (2015) Stac adaptor proteins regulate trafficking and function of muscle and neuronal L-type Ca2+ channels. Proceedings of the National Academy of Sciences of the United States of America. 112: 602-6
Bannister RA, Beam KG. (2013) Impaired gating of an L-Type Ca(2+) channel carrying a mutation linked to malignant hyperthermia. Biophysical Journal. 104: 1917-22
Bannister RA, Beam KG. (2013) Ca(V)1.1: The atypical prototypical voltage-gated Ca²⁺ channel. Biochimica Et Biophysica Acta. 1828: 1587-97
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