Christopher C.J. Miller

Institute of Psychiatry, King's College London Institute of Psychiatry, King's College London, London, England, United Kingdom 
"Christopher Miller"
Mean distance: 22.21 (cluster 50)
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Mórotz GM, Glennon EB, Greig J, et al. (2019) Kinesin light chain-1 serine-460 phosphorylation is altered in Alzheimer's disease and regulates axonal transport and processing of the amyloid precursor protein. Acta Neuropathologica Communications. 7: 200
Mórotz GM, Glennon EB, Gomez-Suaga P, et al. (2019) LMTK2 binds to kinesin light chains to mediate anterograde axonal transport of cdk5/p35 and LMTK2 levels are reduced in Alzheimer's disease brains. Acta Neuropathologica Communications. 7: 73
Paillusson S, Stoica R, Gomez-Suaga P, et al. (2016) There's Something Wrong with my MAM; the ER-Mitochondria Axis and Neurodegenerative Diseases. Trends in Neurosciences
Yuan A, Hassinger L, Rao MV, et al. (2015) Dissociation of Axonal Neurofilament Content from Its Transport Rate. Plos One. 10: e0133848
Chow WN, Ngo JC, Li W, et al. (2015) Phosphorylation of FE65 Serine-610 by serum- and glucocorticoid-induced kinase 1 modulates Alzheimer's disease amyloid precursor protein processing. The Biochemical Journal
Godena VK, Brookes-Hocking N, Moller A, et al. (2014) Increasing microtubule acetylation rescues axonal transport and locomotor deficits caused by LRRK2 Roc-COR domain mutations. Nature Communications. 5: 5245
Stoica R, De Vos KJ, Paillusson S, et al. (2014) ER-mitochondria associations are regulated by the VAPB-PTPIP51 interaction and are disrupted by ALS/FTD-associated TDP-43. Nature Communications. 5: 3996
Cheung HN, Dunbar C, Mórotz GM, et al. (2014) FE65 interacts with ADP-ribosylation factor 6 to promote neurite outgrowth. Faseb Journal : Official Publication of the Federation of American Societies For Experimental Biology. 28: 337-49
Noble W, Hanger DP, Miller CC, et al. (2013) The importance of tau phosphorylation for neurodegenerative diseases. Frontiers in Neurology. 4: 83
Vagnoni A, Glennon EB, Perkinton MS, et al. (2013) Loss of c-Jun N-terminal kinase-interacting protein-1 does not affect axonal transport of the amyloid precursor protein or Aβ production. Human Molecular Genetics. 22: 4646-52
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