Faye Boeckman
Affiliations: | Life Technologies, Carlsbad, CA, United States |
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"Faye Boeckman"Mean distance: 14.66 (cluster 11) | S | N | B | C | P |
Parents
Sign in to add mentor| William A. Catterall | grad student | University of Washington | |
| Elias Aizenman | grad student | 1990-1996 | University of Pittsburgh |
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Publications
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| Rohl CA, Boeckman FA, Baker C, et al. (1999) Solution structure of the sodium channel inactivation gate. Biochemistry. 38: 855-61 |
| Brimecombe JC, Boeckman FA, Aizenman E. (1997) Functional consequences of NR2 subunit composition in single recombinant N-methyl-D-aspartate receptors. Proceedings of the National Academy of Sciences of the United States of America. 94: 11019-24 |
| Blanpied TA, Boeckman FA, Aizenman E, et al. (1997) Trapping channel block of NMDA-activated responses by amantadine and memantine. Journal of Neurophysiology. 77: 309-23 |
| Sinor JD, Boeckman FA, Aizenman E. (1997) Intrinsic redox properties of N-methyl-D-aspartate receptor can determine the developmental expression of excitotoxicity in rat cortical neurons in vitro. Brain Research. 747: 297-303 |
| Boeckman FA, Aizenman E. (1996) Pharmacological properties of acquired excitotoxicity in Chinese hamster ovary cells transfected with N-methyl-D-aspartate receptor subunits. The Journal of Pharmacology and Experimental Therapeutics. 279: 515-23 |
| Boeckman FA, Aizenman E. (1994) Stable transfection of the NR1 subunit in Chinese hamster ovary cells fails to produce a functional N-methyl-D-aspartate receptor. Neuroscience Letters. 173: 189-92 |
| Aizenman E, Boeckman FA, Rosenberg PA. (1992) Glutathione prevents 2,4,5-trihydroxyphenylalanine excitotoxicity by maintaining it in a reduced, non-active form. Neuroscience Letters. 144: 233-6 |