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Matthew P. Frosch, MD PhD

Affiliations: 
Pathology Massachusetts General Hospital and Harvard University, Boston, MA, United States 
Area:
cerebral amyloid angiopathy
Website:
http://hst.mit.edu/public/people/faculty/facultyBiosketch.jsp?key=Frosch
Google:
"Matthew Frosch"
Bio:

http://madrc.mgh.harvard.edu/matthew-p-frosch-md-phd



Mean distance: 16.76 (cluster 24)
 
Cross-listing: Neuropathology Tree - Alzheimer's Tree

BETA: Related publications

Publications

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Mueller C, Berry JD, McKenna-Yasek DM, et al. (2020) Suppression with Adeno-Associated Virus and MicroRNA in Familial ALS. The New England Journal of Medicine. 383: 151-158
Dujardin S, Commins C, Lathuiliere A, et al. (2020) Tau molecular diversity contributes to clinical heterogeneity in Alzheimer's disease. Nature Medicine
Regenhardt RW, Thon JM, Das AS, et al. (2020) Association Between Immunosuppressive Treatment and Outcomes of Cerebral Amyloid Angiopathy-Related Inflammation. Jama Neurology
Oakley DH, Chung M, Klickstein N, et al. (2020) The Alzheimer Disease-Causing Presenilin-1 L435F Mutation Causes Increased Production of Soluble Aβ43 Species in Patient-Derived iPSC-Neurons, Closely Mimicking Matched Patient Brain Tissue. Journal of Neuropathology and Experimental Neurology
Kozberg MG, van Veluw SJ, Frosch MP, et al. (2020) Hereditary cerebral amyloid angiopathy, Piedmont-type mutation. Neurology. Genetics. 6: e411
Ter Telgte A, Scherlek AA, Reijmer YD, et al. (2020) Histopathology of diffusion-weighted imaging-positive lesions in cerebral amyloid angiopathy. Acta Neuropathologica
Reiman EM, Arboleda-Velasquez JF, Quiroz YT, et al. (2020) Exceptionally low likelihood of Alzheimer's dementia in APOE2 homozygotes from a 5,000-person neuropathological study. Nature Communications. 11: 667
van Veluw SJ, Frosch MP, Scherlek AA, et al. (2020) In vivo characterization of spontaneous microhemorrhage formation in mice with cerebral amyloid angiopathy. Journal of Cerebral Blood Flow and Metabolism : Official Journal of the International Society of Cerebral Blood Flow and Metabolism. 271678X19899377
Corbett GT, Wang Z, Hong W, et al. (2019) PrP is a central player in toxicity mediated by soluble aggregates of neurodegeneration-causing proteins. Acta Neuropathologica
van Veluw SJ, Hou SS, Calvo-Rodriguez M, et al. (2019) Vasomotion as a Driving Force for Paravascular Clearance in the Awake Mouse Brain. Neuron
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