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Khalid Iqbal, PhD

Neurochemistry New York State Institute for Basic Research in Disabilities, NY, United States 
"Khalid Iqbal"

Mean distance: 16.44 (cluster 11)
Cross-listing: Alzheimer's Tree - Neuropathology Tree

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Li L, Jiang Y, Hu W, et al. (2019) Pathological Alterations of Tau in Alzheimer's Disease and 3xTg-AD Mouse Brains. Molecular Neurobiology
Gu J, Chen F, Chu D, et al. (2018) Rbfox3/NeuN Regulates Alternative Splicing of Tau Exon 10. Journal of Alzheimer's Disease : Jad
Dai CL, Gu JH, Liu F, et al. (2018) Neuronal O-GlcNAc transferase regulates appetite, body weight, and peripheral insulin resistance. Neurobiology of Aging. 70: 40-50
Hu W, Tung YC, Zhang Y, et al. (2018) Involvement of Activation of Asparaginyl Endopeptidase in Tau Hyperphosphorylation in Repetitive Mild Traumatic Brain Injury. Journal of Alzheimer's Disease : Jad
Basurto-Islas G, Gu JH, Tung YC, et al. (2018) Mechanism of Tau Hyperphosphorylation Involving Lysosomal Enzyme Asparagine Endopeptidase in a Mouse Model of Brain Ischemia. Journal of Alzheimer's Disease : Jad
Gu J, Jin N, Ma D, et al. (2018) Calpain I Activation Causes GLUT3 Proteolysis and Downregulation of O-GlcNAcylation in Alzheimer's Disease Brain. Journal of Alzheimer's Disease : Jad. 62: 1737-1746
Gong CX, Liu F, Iqbal K. (2018) Multifactorial Hypothesis and Multi-Targets for Alzheimer's Disease. Journal of Alzheimer's Disease : Jad
Iqbal K, Liu F, Gong CX. (2018) Recent developments with tau-based drug discovery. Expert Opinion On Drug Discovery. 1-12
Zhou Y, Shi J, Chu D, et al. (2018) Relevance of Phosphorylation and Truncation of Tau to the Etiopathogenesis of Alzheimer's Disease. Frontiers in Aging Neuroscience. 10: 27
Jin N, Ma D, Gu J, et al. (2018) O-GlcNAcylation modulates PKA-CREB signaling in a manner specific to PKA catalytic subunit isoforms. Biochemical and Biophysical Research Communications
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