Joel D. Schilling, Ph.D.
Affiliations: | 2003 | Washington University, Saint Louis, St. Louis, MO |
Area:
Immunology, Microbiology Biology, PathologyGoogle:
"Joel Schilling"Parents
Sign in to add mentorScott J. Hultgren | grad student | 2003 | Washington University | |
(The dynamic interaction between uropathogenic E. coli and the bladder mucosa.) |
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Publications
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Chandra P, He L, Zimmerman M, et al. (2020) Inhibition of Fatty Acid Oxidation Promotes Macrophage Control of Mycobacterium tuberculosis. Mbio. 11 |
Javaheri A, Bajpai G, Picataggi A, et al. (2019) TFEB activation in macrophages attenuates postmyocardial infarction ventricular dysfunction independently of ATG5-mediated autophagy. Jci Insight. 4 |
Kalugotla G, He L, Weber KJ, et al. (2019) Frontline Science: Acyl-CoA synthetase 1 exacerbates lipotoxic inflammasome activation in primary macrophages. Journal of Leukocyte Biology |
Weber KJ, Sauer M, He L, et al. (2018) PPARγ Deficiency Suppresses the Release of IL-1β and IL-1α in Macrophages via a Type 1 IFN-Dependent Mechanism. Journal of Immunology (Baltimore, Md. : 1950) |
Sergin I, Evans TD, Zhang X, et al. (2017) Exploiting macrophage autophagy-lysosomal biogenesis as a therapy for atherosclerosis. Nature Communications. 8: 15750 |
Artyomov MN, Sergushichev A, Schilling JD. (2016) Integrating immunometabolism and macrophage diversity. Seminars in Immunology |
He L, Weber KJ, Diwan A, et al. (2016) Inhibition of mTOR reduces lipotoxic cell death in primary macrophages through an autophagy-independent mechanism. Journal of Leukocyte Biology |
He L, Weber KJ, Schilling JD. (2016) Glutamine Modulates Macrophage Lipotoxicity. Nutrients. 8 |
Weber K, Schilling JD. (2014) Distinct lysosome phenotypes influence inflammatory function in peritoneal and bone marrow-derived macrophages. International Journal of Inflammation. 2014: 154936 |
Lavine KJ, Epelman S, Uchida K, et al. (2014) Distinct macrophage lineages contribute to disparate patterns of cardiac recovery and remodeling in the neonatal and adult heart. Proceedings of the National Academy of Sciences of the United States of America. 111: 16029-34 |