Diego Sepulveda-Falla, M.D.

Affiliations: 
Institute of Neuropathology UKE Hamburg 
Area:
Alzheimer
Google:
"Diego Sepulveda-Falla"
Mean distance: 23.53 (cluster 33)
 
Cross-listing: Neuropathology Tree - Alzheimer's Tree

BETA: Related publications

Publications

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Podlesniy P, Llorens F, Puigròs M, et al. (2020) Cerebrospinal Fluid Mitochondrial DNA in Rapid and Slow Progressive Forms of Alzheimer's Disease. International Journal of Molecular Sciences. 21
Dinkel F, Trujillo-Rodriguez D, Villegas A, et al. (2020) Decreased Deposition of Beta-Amyloid 1-38 and Increased Deposition of Beta-Amyloid 1-42 in Brain Tissue of Presenilin-1 E280A Familial Alzheimer's Disease Patients. Frontiers in Aging Neuroscience. 12: 220
Palmqvist S, Janelidze S, Quiroz YT, et al. (2020) Discriminative Accuracy of Plasma Phospho-tau217 for Alzheimer Disease vs Other Neurodegenerative Disorders. Jama
Quiroz YT, Zetterberg H, Reiman EM, et al. (2020) Plasma neurofilament light chain in the presenilin 1 E280A autosomal dominant Alzheimer's disease kindred: a cross-sectional and longitudinal cohort study. The Lancet. Neurology. 19: 513-521
Sabogal-Guáqueta AM, Arias-Londoño JD, Gutierrez-Vargas J, et al. (2020) Common disbalance in the brain parenchyma of dementias: Phospholipid profile analysis between CADASIL and sporadic Alzheimer's disease. Biochimica Et Biophysica Acta. Molecular Basis of Disease. 1866: 165797
Rojas-Charry L, Calero-Martinez S, Morganti C, et al. (2020) Susceptibility to cellular stress in PS1 mutant N2a cells is associated with mitochondrial defects and altered calcium homeostasis. Scientific Reports. 10: 6455
Hermann M, Reumann R, Schostak K, et al. (2019) Deficits in developmental neurogenesis and dendritic spine maturation in mice lacking the serine protease inhibitor neuroserpin. Molecular and Cellular Neurosciences. 102: 103420
Muth C, Hartmann A, Sepulveda-Falla D, et al. (2019) Phagocytosis of Apoptotic Cells Is Specifically Upregulated in ApoE4 Expressing Microglia . Frontiers in Cellular Neuroscience. 13: 181
Hartmann K, Sepulveda-Falla D, Rose IVL, et al. (2019) Complement 3-astrocytes are highly abundant in prion diseases, but their abolishment led to an accelerated disease course and early dysregulation of microglia. Acta Neuropathologica Communications. 7: 83
Arcos-Burgos M, Lopera F, Sepulveda-Falla D, et al. (2019) Neural Plasticity during Aging. Neural Plasticity. 2019: 6042132
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