Marco Sardiello
Affiliations: | Genetics | Telethon Institute of Genetics and Medicine, Italy |
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Parents
Sign in to add mentor| Andrea Ballabio | post-doc | 2003-2010 | Telethon Institute of Genetics and Medicine |
Children
Sign in to add trainee| Kevin Tommy Chang | research assistant | 2015-2020 | Baylor College of Medicine |
| Lakshya Bajaj | grad student | 2012-2018 | Baylor College of Medicine |
BETA: Related publications
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Publications
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| Sharma J, Mulherkar S, Chen UI, et al. (2023) Calpain activity is negatively regulated by a KCTD7-Cullin-3 complex via non-degradative ubiquitination. Cell Discovery. 9: 32 |
| Klionsky DJ, Abdel-Aziz AK, Abdelfatah S, et al. (2021) Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition). Autophagy. 1-382 |
| Bajaj L, Sharma J, di Ronza A, et al. (2020) A CLN6-CLN8 complex recruits lysosomal enzymes at the ER for Golgi transfer. The Journal of Clinical Investigation |
| Mao D, Lin G, Tepe B, et al. (2019) VAMP associated proteins are required for autophagic and lysosomal degradation by promoting a PtdIns4P-mediated endosomal pathway. Autophagy |
| Pal R, Xiong Y, Sardiello M. (2019) Abnormal glycogen storage in tuberous sclerosis complex caused by impairment of mTORC1-dependent and -independent signaling pathways. Proceedings of the National Academy of Sciences of the United States of America |
| di Ronza A, Bajaj L, Sharma J, et al. (2018) CLN8 is an endoplasmic reticulum cargo receptor that regulates lysosome biogenesis. Nature Cell Biology |
| Pal R, Palmieri M, Chaudhury A, et al. (2018) Src regulates amino acid-mediated mTORC1 activation by disrupting GATOR1-Rag GTPase interaction. Nature Communications. 9: 4351 |
| Martini-Stoica H, Cole AL, Swartzlander DB, et al. (2018) TFEB enhances astroglial uptake of extracellular tau species and reduces tau spreading. The Journal of Experimental Medicine |
| Bajaj L, Lotfi P, Pal R, et al. (2018) Lysosome biogenesis in health and disease. Journal of Neurochemistry |
| Tokita MJ, Chen CA, Chitayat D, et al. (2018) De Novo Missense Variants in TRAF7 Cause Developmental Delay, Congenital Anomalies, and Dysmorphic Features. American Journal of Human Genetics |