Robert J. Connacher
Affiliations: | Neuroscience & Cell Biology | Rutgers University, New Brunswick, New Brunswick, NJ, United States |
Area:
Developmental NeuroscienceGoogle:
"Robert Connacher"Mean distance: 17.48 (cluster 6)
Parents
Sign in to add mentor| Emanuel M. DiCicco-Bloom | grad student | 2011- | UMDNJ-Robert Wood Johnson Medical School |
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Publications
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| Prem S, Dev B, Peng C, et al. (2024) Dysregulation of mTOR signaling mediates common neurite and migration defects in both idiopathic and 16p11.2 deletion autism neural precursor cells. Elife. 13 |
| Connacher R, Williams M, Prem S, et al. (2022) Autism NPCs from both idiopathic and CNV 16p11.2 deletion patients exhibit dysregulation of proliferation and mitogenic responses. Stem Cell Reports. 17: 1786 |
| Connacher R, Williams M, Prem S, et al. (2022) Autism NPCs from both idiopathic and CNV 16p11.2 deletion patients exhibit dysregulation of proliferation and mitogenic responses. Stem Cell Reports |
| Turkalj L, Mehta M, Matteson P, et al. (2020) Using iPSC-Based Models to Understand the Signaling and Cellular Phenotypes in Idiopathic Autism and 16p11.2 Derived Neurons. Advances in Neurobiology. 25: 79-107 |
| Connacher RJ, DiCicco-Bloom E, Millonig JH. (2018) Using Human Induced Neural Precursor Cells to Define Early Neurodevelopmental Defects in Syndromic and Idiopathic Autism Current Pharmacology Reports. 4: 422-435 |