Koji Yamanaka
Affiliations: | Riken Brain Institute, Japan |
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| Horiuchi M, Watanabe S, Komine O, et al. (2024) ALS-linked mutant TDP-43 in oligodendrocytes induces oligodendrocyte damage and exacerbates motor dysfunction in mice. Acta Neuropathologica Communications. 12: 184 |
| Watanabe S, Amporndanai K, Awais R, et al. (2024) Ebselen analogues delay disease onset and its course in fALS by on-target SOD-1 engagement. Scientific Reports. 14: 12118 |
| Leventoux N, Morimoto S, Ishikawa M, et al. (2024) Aberrant CHCHD2-associated mitochondriopathy in Kii ALS/PDC astrocytes. Acta Neuropathologica. 147: 84 |
| Komine O, Ohnuma S, Hinohara K, et al. (2024) Genetic background variation impacts microglial heterogeneity and disease progression in amyotrophic lateral sclerosis model mice. Iscience. 27: 108872 |
| Adachi K, Miyata K, Chida Y, et al. (2023) Depletion of perivascular macrophages delays ALS disease progression by ameliorating blood-spinal cord barrier impairment in SOD1 mice. Frontiers in Cellular Neuroscience. 17: 1291673 |
| Oiwa K, Watanabe S, Onodera K, et al. (2023) Monomerization of TDP-43 is a key determinant for inducing TDP-43 pathology in amyotrophic lateral sclerosis. Science Advances. 9: eadf6895 |
| Yamashita H, Komine O, Fujimori-Tonou N, et al. (2023) Corrigendum: Comprehensive expression analysis with cell-type-specific transcriptome in ALS-linked mutant SOD1 mice: Revisiting the active role of glial cells in disease. Frontiers in Cellular Neuroscience. 17: 1160444 |
| Yamashita H, Komine O, Fujimori-Tonou N, et al. (2023) Comprehensive expression analysis with cell-type-specific transcriptome in ALS-linked mutant SOD1 mice: Revisiting the active role of glial cells in disease. Frontiers in Cellular Neuroscience. 16: 1045647 |
| Sakai S, Watanabe S, Komine O, et al. (2021) Novel reporters of mitochondria-associated membranes (MAM), MAMtrackers, demonstrate MAM disruption as a common pathological feature in amyotrophic lateral sclerosis. Faseb Journal : Official Publication of the Federation of American Societies For Experimental Biology. 35: e21688 |
| Konishi H, Okamoto T, Hara Y, et al. (2020) Astrocytic phagocytosis is a compensatory mechanism for microglial dysfunction. The Embo Journal. e104464 |