Terina N. Martinez, PhD
Affiliations: | Michael J Fox Foundation |
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"Terina Martinez"Mean distance: 17.21 (cluster 32) | S | N | B | C | P |
Parents
Sign in to add mentor| Malu G Tansey | grad student | 2005-2010 | HHMI/UT Southwestern Medical Center |
| J. Timothy Greenamyre | post-doc | 2010-2013 | University of Pittsburgh |
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Publications
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| Polinski NK, Martinez TN, Ramboz S, et al. (2022) The GBA1 D409V mutation exacerbates synuclein pathology to differing extents in two alpha-synuclein models. Disease Models & Mechanisms |
| Polinski NK, Martinez TN, Gorodinsky A, et al. (2021) Decreased glucocerebrosidase activity and substrate accumulation of glycosphingolipids in a novel GBA1 D409V knock-in mouse model. Plos One. 16: e0252325 |
| Mir R, Tonelli F, Lis P, et al. (2018) The Parkinson's disease VPS35[D620N] mutation enhances LRRK2 mediated Rab protein phosphorylation in mouse and human. The Biochemical Journal |
| Polinski NK, Volpicelli-Daley LA, Sortwell CE, et al. (2018) Best Practices for Generating and Using Alpha-Synuclein Pre-Formed Fibrils to Model Parkinson's Disease in Rodents. Journal of Parkinson's Disease |
| Purlyte E, Dhekne HS, Sarhan AR, et al. (2017) Rab29 activation of the Parkinson's disease-associated LRRK2 kinase. The Embo Journal |
| Fan Y, Howden AJ, Sarhan AR, et al. (2017) Interrogating Parkinson's disease LRRK2 kinase pathway activity by assessing Rab10 phosphorylation in human neutrophils. The Biochemical Journal |
| Steger M, Diez F, Dhekne HS, et al. (2017) Systematic proteomic analysis of LRRK2-mediated Rab GTPase phosphorylation establishes a connection to ciliogenesis. Elife. 6 |
| Iacovitti L, Wei X, Cai J, et al. (2014) The hTH-GFP reporter rat model for the study of Parkinson's disease. Plos One. 9: e113151 |
| Nguyen TA, Frank-Cannon T, Martinez TN, et al. (2013) Analysis of inflammation-related nigral degeneration and locomotor function in DJ-1(-/-) mice. Journal of Neuroinflammation. 10: 50 |
| Martinez TN, Chen X, Bandyopadhyay S, et al. (2012) Ceramide sphingolipid signaling mediates Tumor Necrosis Factor (TNF)-dependent toxicity via caspase signaling in dopaminergic neurons. Molecular Neurodegeneration. 7: 45 |