Andrew N. Blackford, Ph.D.

Affiliations: 
University of Oxford, Oxford, United Kingdom 
Area:
DNA damage response
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Parents

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Roger J. Grand grad student (Chemistry Tree)
Stephen P. Jackson post-doc
Wojciech Niedzwiedz post-doc
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Publications

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Tsukada K, Jones SE, Bannister J, et al. (2024) BLM and BRCA1-BARD1 coordinate complementary mechanisms of joint DNA molecule resolution. Molecular Cell
Groelly FJ, Fawkes M, Dagg RA, et al. (2022) Targeting DNA damage response pathways in cancer. Nature Reviews. Cancer. 23: 78-94
Groelly FJ, Dagg RA, Petropoulos M, et al. (2022) Mitotic DNA synthesis is caused by transcription-replication conflicts in BRCA2-deficient cells. Molecular Cell
De Marco Zompit M, Esteban MT, Mooser C, et al. (2022) The CIP2A-TOPBP1 complex safeguards chromosomal stability during mitosis. Nature Communications. 13: 4143
Shorrocks AK, Jones SE, Tsukada K, et al. (2021) The Bloom syndrome complex senses RPA-coated single-stranded DNA to restart stalled replication forks. Nature Communications. 12: 585
Henssen AG, Koche R, Zhuang J, et al. (2020) Author Correction: PGBD5 promotes site-specific oncogenic mutations in human tumors. Nature Genetics
Ochi T, Quarantotti V, Lin H, et al. (2020) CCDC61/VFL3 Is a Paralog of SAS6 and Promotes Ciliary Functions. Structure (London, England : 1993)
Blackford AN, Stucki M. (2020) How Cells Respond to DNA Breaks in Mitosis. Trends in Biochemical Sciences
Mooser C, Symeonidou IE, Leimbacher PA, et al. (2020) Treacle controls the nucleolar response to rDNA breaks via TOPBP1 recruitment and ATR activation. Nature Communications. 11: 123
Leimbacher PA, Jones SE, Shorrocks AK, et al. (2019) MDC1 Interacts with TOPBP1 to Maintain Chromosomal Stability during Mitosis. Molecular Cell
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