Marshall Lukacs
Affiliations: | Cincinnati Children's Hospital Medical Center, University of Cincinnati |
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| Abdelhamed Z, Lukacs M, Cindric S, et al. (2021) Correction: A novel hypomorphic allele of Spag17 causes primary ciliary dyskinesia phenotypes in mice. Disease Models & Mechanisms. 14 |
| Abdelhamed Z, Lukacs M, Cindric S, et al. (2021) Correction: A novel hypomorphic allele of Spag17 causes primary ciliary dyskinesia phenotypes in mice Disease Models & Mechanisms. 14: dmm048645 |
| Abdelhamed Z, Lukacs M, Cindric S, et al. (2020) A novel hypomorphic allele of Spag17 causes primary ciliary dyskinesia phenotypes in mice. Disease Models & Mechanisms |
| Abdelhamed Z, Lukacs M, Cindric S, et al. (2020) A novel hypomorphic allele of causes primary ciliary dyskinesia phenotypes in mice. Disease Models & Mechanisms |
| Lukacs M, Blizzard LE, Stottmann RW. (2020) CNS Glycosylphosphatidylinositol Deficiency Results in Delayed White Matter Development, Ataxia, and Premature Death in a Novel Mouse Model. Human Molecular Genetics |
| Lukacs M, Roberts T, Chatuverdi P, et al. (2019) Glycosylphosphatidylinositol biosynthesis and remodeling are required for neural tube closure, heart development, and cranial neural crest cell survival. Elife. 8 |
| Lukacs M, Gilley J, Zhu Y, et al. (2019) Severe biallelic loss-of-function mutations in nicotinamide mononucleotide adenylyltransferase 2 (NMNAT2) in two fetuses with fetal akinesia deformation sequence. Experimental Neurology. 112961 |
| Gelineau-Morel R, Lukacs M, Weaver KN, et al. (2016) Congenital Cataracts and Gut Dysmotility in a DYNC1H1 Dyneinopathy Patient. Genes. 7 |