Kyoung-Han Kim
Affiliations: | University of Ottawa, Ottawa, ON, Canada |
Area:
Development and PhysiologyGoogle:
"Kyoung-Han Kim"
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Publications
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| Lu A, Wang J, Xia Y, et al. (2022) Viral Transgene Expression in Rodent Hearts and the Assessment of Cardiac Arrhythmia Risk. Journal of Visualized Experiments : Jove |
| Kim KH, Oh Y, Liu J, et al. (2022) Irx5 and transient outward K currents contribute to transmural contractile heterogeneities in the mouse ventricle. American Journal of Physiology. Heart and Circulatory Physiology. 322: H725-H741 |
| Kim KH, Backx PH. (2021) Understanding the Role of Iroquois Homeobox Transcription Factor 5 (IRX5) in Cardiac Function: Getting to the (Human) Heart of the Matter. Cardiovascular Research |
| Oh Y, Yang S, Liu X, et al. (2020) Transcriptomic Bioinformatic Analyses of Atria Uncover Involvement of Pathways Related to Strain and Post-translational Modification of Collagen in Increased Atrial Fibrillation Vulnerability in Intensely Exercised Mice. Frontiers in Physiology. 11: 605671 |
| Lin HB, Naito K, Oh Y, et al. (2020) Innate Immune Nod1/RIP2 Signaling is Essential for Cardiac Hypertrophy, but Requires Mitochondrial Antiviral Signaling Protein (MAVS) for Signal Transductions and Energy Balance. Circulation |
| Kim KH, Rosen A, Hussein SM, et al. (2016) Irx3 is required for postnatal maturation of the mouse ventricular conduction system. Scientific Reports. 6: 19197 |
| Cordeiro JM, Calloe K, Aschar-Sobbi R, et al. (2016) Physiological roles of the transient outward current Ito in normal and diseased hearts. Frontiers in Bioscience (Scholar Edition). 8: 143-59 |
| Liu J, Kim KH, Morales MJ, et al. (2015) Kv4.3-Encoded Fast Transient Outward Current Is Presented in Kv4.2 Knockout Mouse Cardiomyocytes. Plos One. 10: e0133274 |
| Gaborit N, Sakuma R, Wylie JN, et al. (2012) Cooperative and antagonistic roles for Irx3 and Irx5 in cardiac morphogenesis and postnatal physiology. Development (Cambridge, England). 139: 4007-19 |
| Wu X, Yin J, Simpson J, et al. (2012) Increased BRAF heterodimerization is the common pathogenic mechanism for noonan syndrome-associated RAF1 mutants. Molecular and Cellular Biology. 32: 3872-90 |