James S. Malter, MD

Affiliations: 
1991-2011 University of Wisconsin, Madison, Madison, WI 
 2011- Pathology University of Texas Southwestern Medical Center, Dallas, TX, United States 
Area:
RNA biology, Alzheimers Disese
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"James Malter"
Mean distance: 17.73 (cluster 6)
 

Children

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Peter Todd grad student 1999-2003 UW Madison
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Publications

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Shen ZJ, Hu J, Kashi V, et al. (2018) TLR-7 Stress Signaling in Differentiating and Mature Eosinophils Is Mediated by the Prolyl Isomerase Pin1. Journal of Immunology (Baltimore, Md. : 1950)
Esnault S, Shen ZJ, Malter JS. (2017) Protein Translation and Signaling in Human Eosinophils. Frontiers in Medicine. 4: 150
Shen ZJ, Hu J, Kashi V, et al. (2017) Epstein-Barr Virus Induced Gene 2 (EBI2) Mediates Allergen-induced, Leukocyte Migration into Airways. American Journal of Respiratory and Critical Care Medicine
Kelly EA, Esnault S, Johnson SH, et al. (2016) Human eosinophil activin A synthesis and mRNA stabilization are induced by the combination of IL-3 plus TNF. Immunology and Cell Biology
Esnault S, Kelly EA, Shen ZJ, et al. (2015) IL-3 Maintains Activation of the p90S6K/RPS6 Pathway and Increases Translation in Human Eosinophils. Journal of Immunology (Baltimore, Md. : 1950). 195: 2529-39
Shen ZJ, Hu J, Esnault S, et al. (2015) RNA Seq profiling reveals a novel expression pattern of TGF-β target genes in human blood eosinophils. Immunology Letters
Shen ZJ, Malter JS. (2015) Determinants of eosinophil survival and apoptotic cell death. Apoptosis : An International Journal On Programmed Cell Death. 20: 224-34
Esnault S, Kelly EA, Johansson MW, et al. (2014) Semaphorin 7A is expressed on airway eosinophils and upregulated by IL-5 family cytokines. Clinical Immunology (Orlando, Fla.). 150: 90-100
Oh J, Malter JS. (2013) Pin1-FADD interactions regulate fas-mediated apoptosis in activated eosinophils Journal of Immunology. 190: 4937-4945
Shen ZJ, Braun RK, Hu J, et al. (2012) Pin1 protein regulates smad protein signaling and pulmonary fibrosis Journal of Biological Chemistry. 287: 23294-23305
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