Kyle J. Eash, Ph.D. - Publications
Affiliations: | 2011 | Biology & Biomedical Sciences (Molecular Cell Biology) | Washington University, Saint Louis, St. Louis, MO |
Area:
Immunology, Medicine and Surgery, Animal Physiology BiologyYear | Citation | Score | |||
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2010 | Eash KJ, Greenbaum AM, Gopalan PK, Link DC. CXCR2 and CXCR4 antagonistically regulate neutrophil trafficking from murine bone marrow. The Journal of Clinical Investigation. 120: 2423-31. PMID 20516641 DOI: 10.1172/Jci41649 | 0.629 | |||
2009 | Eash KJ, Means JM, White DW, Link DC. CXCR4 is a key regulator of neutrophil release from the bone marrow under basal and stress granulopoiesis conditions. Blood. 113: 4711-9. PMID 19264920 DOI: 10.1182/Blood-2008-09-177287 | 0.77 | |||
2009 | Eash KJ, Greenbaum AM, Gopalan P, Diaz GA, Link DC. CXCR2 Signals Act in Concert with CXCR4 to Regulate Neutrophil Release From the Bone Marrow. Blood. 114: 235-235. DOI: 10.1182/Blood.V114.22.235.235 | 0.639 | |||
2007 | Eash KJ, Means JM, Woloszynek JR, Liu F, Link DC. CXCR4 Signals Regulate Neutrophil Release from the Bone Marrow but Not Clearance from the Circulation. Blood. 110: 3297-3297. DOI: 10.1182/Blood.V110.11.3297.3297 | 0.776 | |||
2006 | Eash KJ, Link DC. CXCR4 Signals Regulate Basal and G-CSF Induced Neutrophil Release from the Bone Marrow. Blood. 108: 673-673. DOI: 10.1182/Blood.V108.11.673.673 | 0.754 | |||
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