☰

Ge Bai, Ph.D. - Publications

Affiliations: 
Salk Institute for Biological Studies, La Jolla, CA, United States 
Tree Info Grants Similar researchers PubMed Report error

14 high-probability publications. We are testing a new system for linking publications to authors. You can help! If you notice any inaccuracies, please sign in and mark papers as correct or incorrect matches. If you identify any major omissions or other inaccuracies in the publication list, please let us know.

Year Citation  Score
2023 Liu H, Tang M, Yu H, Liu T, Cui Q, Gu L, Wu Z, Sheng N, Yang XL, Zeng L, Bai G. CMT2D neuropathy is influenced by vitamin D-mediated environmental pathway. Journal of Molecular Cell Biology. PMID 37118880 DOI: 10.1093/jmcb/mjad029  0.438
2023 Cui Q, Bi H, Lv Z, Wu Q, Hua J, Gu B, Huo C, Tang M, Chen Y, Chen C, Chen S, Zhang X, Wu Z, Lao Z, Sheng N, ... ... Bai G, et al. Diverse CMT2 neuropathies are linked to aberrant G3BP interactions in stress granules. Cell. PMID 36738734 DOI: 10.1016/j.cell.2022.12.046  0.628
2018 Mo Z, Zhao X, Liu H, Hu Q, Chen XQ, Pham J, Wei N, Liu Z, Zhou J, Burgess RW, Pfaff SL, Caskey CT, Wu C, Bai G, Yang XL. Aberrant GlyRS-HDAC6 interaction linked to axonal transport deficits in Charcot-Marie-Tooth neuropathy. Nature Communications. 9: 1007. PMID 29520015 DOI: 10.1038/S41467-018-03461-Z  0.608
2017 Sleigh JN, Gómez-Martín A, Wei N, Bai G, Yang XL, Schiavo G. Neuropilin 1 sequestration by neuropathogenic mutant glycyl-tRNA synthetase is permissive to vascular homeostasis. Scientific Reports. 7: 9216. PMID 28835631 DOI: 10.1038/S41598-017-10005-W  0.303
2016 He W, Bai G, Zhou H, Wei N, White NM, Lauer J, Liu H, Shi Y, Dan Dumitru C, Lettieri K, Shubayev V, Jordanova A, Guergueltcheva V, Griffin PR, Burgess RW, et al. Corrigendum: CMT2D neuropathy is linked to the neomorphic binding activity of glycyl-tRNA synthetase. Nature. PMID 26789244 DOI: 10.1038/Nature16499  0.53
2015 Amin ND, Bai G, Klug JR, Bonanomi D, Pankratz MT, Gifford WD, Hinckley CA, Sternfeld MJ, Driscoll SP, Dominguez B, Lee KF, Jin X, Pfaff SL. Loss of motoneuron-specific microRNA-218 causes systemic neuromuscular failure. Science (New York, N.Y.). 350: 1525-9. PMID 26680198 DOI: 10.1126/Science.Aad2509  0.566
2015 He W, Bai G, Zhou H, Wei N, White NM, Lauer J, Liu H, Shi Y, Dumitru CD, Lettieri K, Shubayev V, Jordanova A, Guergueltcheva V, Griffin PR, Burgess RW, et al. CMT2D neuropathy is linked to the neomorphic binding activity of glycyl-tRNA synthetase. Nature. 526: 710-4. PMID 26503042 DOI: 10.1038/Nature15510  0.63
2012 Bonanomi D, Chivatakarn O, Bai G, Abdesselem H, Lettieri K, Marquardt T, Pierchala BA, Pfaff SL. Ret is a multifunctional coreceptor that integrates diffusible- and contact-axon guidance signals. Cell. 148: 568-82. PMID 22304922 DOI: 10.1016/J.Cell.2012.01.024  0.702
2011 Bai G, Pfaff SL. Protease regulation: the Yin and Yang of neural development and disease. Neuron. 72: 9-21. PMID 21982365 DOI: 10.1016/J.Neuron.2011.09.012  0.614
2011 Xie Z, Chen Y, Li Z, Bai G, Zhu Y, Yan R, Tan F, Chen YG, Guillemot F, Li L, Jing N. Smad6 promotes neuronal differentiation in the intermediate zone of the dorsal neural tube by inhibition of the Wnt/beta-catenin pathway. Proceedings of the National Academy of Sciences of the United States of America. 108: 12119-24. PMID 21730158 DOI: 10.1073/Pnas.1100160108  0.62
2011 Bai G, Chivatakarn O, Bonanomi D, Lettieri K, Franco L, Xia C, Stein E, Ma L, Lewcock JW, Pfaff SL. Presenilin-dependent receptor processing is required for axon guidance. Cell. 144: 106-18. PMID 21215373 DOI: 10.1016/J.Cell.2010.11.053  0.742
2010 Sheng N, Xie Z, Wang C, Bai G, Zhang K, Zhu Q, Song J, Guillemot F, Chen YG, Lin A, Jing N. Retinoic acid regulates bone morphogenic protein signal duration by promoting the degradation of phosphorylated Smad1. Proceedings of the National Academy of Sciences of the United States of America. 107: 18886-91. PMID 20956305 DOI: 10.1073/Pnas.1009244107  0.665
2008 Bai G, Sheng N, Xie Z, Bian W, Yokota Y, Benezra R, Kageyama R, Guillemot F, Jing N. Id proteins sustain Hes1 expression to maintain neural stem cells Cell Research. 18: S19-S19. DOI: 10.1038/Cr.2008.109  0.643
2007 Bai G, Sheng N, Xie Z, Bian W, Yokota Y, Benezra R, Kageyama R, Guillemot F, Jing N. Id sustains Hes1 expression to inhibit precocious neurogenesis by releasing negative autoregulation of Hes1. Developmental Cell. 13: 283-97. PMID 17681138 DOI: 10.1016/J.Devcel.2007.05.014  0.678
Show low-probability matches.