Nasser Zawia - Publications

Affiliations: 
University of Rhode Island, Providence, RI, United States 
Area:
Toxicology, Neuroscience Biology, Developmental Psychology

81 high-probability publications. We are testing a new system for linking publications to authors. You can help! If you notice any inaccuracies, please sign in and mark papers as correct or incorrect matches. If you identify any major omissions or other inaccuracies in the publication list, please let us know.

Year Citation  Score
2019 Leso A, Bihaqi SW, Masoud A, Chang JK, Lahouel A, Zawia N. Loss in efficacy measures of tolfenamic acid in a tau knock-out model: Relevance to Alzheimer's disease. Experimental Biology and Medicine (Maywood, N.J.). 1535370219871249. PMID 31450960 DOI: 10.1177/1535370219871249  0.44
2018 Bihaqi SW, Alansi B, Masoud AM, Mushtaq F, Subaiea GM, Zawia NH. Influence of Early Life Lead (Pb) exposure on α-Synuclein, GSK-3β and Caspase-3 mediated Tauopathy: Implications on Alzheimer's disease. Current Alzheimer Research. PMID 30068273 DOI: 10.2174/1567205015666180801095925  0.64
2018 Eid A, Bihaqi SW, Hemme C, Gaspar JM, Hart RP, Zawia NH. Histone acetylation maps in aged mice developmentally exposed to lead: epigenetic drift and Alzheimer-related genes. Epigenomics. PMID 29722544 DOI: 10.2217/epi-2017-0143  0.44
2018 Masoud AM, Bihaqi SW, Alansi B, Dash M, Subaiea GM, Renehan WE, Zawia NH. Altered microRNA, mRNA, and Protein Expression of Neurodegeneration-Related Biomarkers and Their Transcriptional and Epigenetic Modifiers in a Human Tau Transgenic Mouse Model in Response to Developmental Lead Exposure. Journal of Alzheimer's Disease : Jad. PMID 29614648 DOI: 10.3233/JAD-170824  0.64
2018 Chang JK, Leso A, Subaiea GM, Lahouel A, Masoud A, Mushtaq F, Deeb R, Eid A, Dash M, Bihaqi SW, Zawia NH. Tolfenamic acid: A modifier of the tau protein and its role in cognition and tauopathy. Current Alzheimer Research. PMID 29357795 DOI: 10.2174/1567205015666180119104036  0.64
2017 Bihaqi SW, Eid A, Zawia NH. Lead exposure and tau hyperphosphorylation: An in-Vitro study. Neurotoxicology. 62: 218-223. PMID 28765091 DOI: 10.1016/j.neuro.2017.07.029  0.44
2016 Dash M, Eid A, Subaiea G, Chang J, Deeb R, Masoud A, Renehan WE, Adem A, Zawia NH. Developmental exposure to lead (Pb) alters the expression of the Human Tau Gene and its products in a transgenic animal model. Neurotoxicology. PMID 27293183 DOI: 10.1016/j.neuro.2016.06.001  0.64
2016 Eid A, Bihaqi SW, Renehan WE, Zawia NH. Developmental lead exposure and lifespan alterations in epigenetic regulators and their correspondence to biomarkers of Alzheimer's disease. Alzheimer's & Dementia (Amsterdam, Netherlands). 2: 123-31. PMID 27239543 DOI: 10.1016/j.dadm.2016.02.002  0.64
2016 Eid A, Zawia N. Consequences of lead exposure, and it's emerging role as an epigenetic modifier in the aging brain. Neurotoxicology. PMID 27066759 DOI: 10.1016/j.neuro.2016.04.006  0.64
2016 Masoud AM, Bihaqi SW, Machan JT, Zawia NH, Renehan WE. Early-Life Exposure to Lead (Pb) Alters the Expression of microRNA that Target Proteins Associated with Alzheimer's Disease. Journal of Alzheimer's Disease : Jad. PMID 26923026 DOI: 10.3233/JAD-151018  0.64
2015 Adwan L, Subaiea GM, Basha R, Zawia NH. Tolfenamic acid reduces tau and CDK5 levels: implications for dementia and tauopathies. Journal of Neurochemistry. 133: 266-72. PMID 25279694 DOI: 10.1111/jnc.12960  0.64
2015 Subaiea GM, Ahmed AH, Adwan LI, Zawia NH. Reduction of amyloid-β deposition and attenuation of memory deficits by tolfenamic acid. Journal of Alzheimer's Disease : Jad. 43: 425-33. PMID 25096618 DOI: 10.3233/JAD-132726  0.64
2014 Ahmed AH, Subaiea GM, Eid A, Li L, Seeram NP, Zawia NH. Pomegranate extract modulates processing of amyloid-β precursor protein in an aged Alzheimer's disease animal model. Current Alzheimer Research. 11: 834-43. PMID 25274111  0.64
2014 Bihaqi SW, Bahmani A, Adem A, Zawia NH. Infantile postnatal exposure to lead (Pb) enhances tau expression in the cerebral cortex of aged mice: relevance to AD. Neurotoxicology. 44: 114-20. PMID 24954411 DOI: 10.1016/j.neuro.2014.06.008  0.64
2014 Adwan L, Subaiea GM, Zawia NH. Tolfenamic acid downregulates BACE1 and protects against lead-induced upregulation of Alzheimer's disease related biomarkers. Neuropharmacology. 79: 596-602. PMID 24462621 DOI: 10.1016/j.neuropharm.2014.01.009  0.64
2014 Bihaqi SW, Bahmani A, Subaiea GM, Zawia NH. Infantile exposure to lead and late-age cognitive decline: relevance to AD. Alzheimer's & Dementia : the Journal of the Alzheimer's Association. 10: 187-95. PMID 23867794 DOI: 10.1016/j.jalz.2013.02.012  0.64
2014 Ahmed AH, Subaiea GM, Eid A, Li L, Seeram NP, Zawia NH. Pomegranate extract modulates processing of amyloid-β precursor protein in an aged Alzheimer’s disease animal model Current Alzheimer Research. 11: 834-843.  0.64
2013 Sankpal UT, Lee CM, Connelly SF, Kayaleh O, Eslin D, Sutphin R, Goodison S, Adwan L, Zawia NH, Lichtenberger LM, Basha R. Cellular and organismal toxicity of the anti-cancer small molecule, tolfenamic acid: a pre-clinical evaluation. Cellular Physiology and Biochemistry : International Journal of Experimental Cellular Physiology, Biochemistry, and Pharmacology. 32: 675-86. PMID 24030139 DOI: 10.1159/000354471  0.64
2013 Bihaqi SW, Zawia NH. Enhanced taupathy and AD-like pathology in aged primate brains decades after infantile exposure to lead (Pb). Neurotoxicology. 39: 95-101. PMID 23973560 DOI: 10.1016/j.neuro.2013.07.010  0.64
2013 Subaiea GM, Adwan LI, Ahmed AH, Stevens KE, Zawia NH. Short-term treatment with tolfenamic acid improves cognitive functions in Alzheimer's disease mice. Neurobiology of Aging. 34: 2421-30. PMID 23639209 DOI: 10.1016/j.neurobiolaging.2013.04.002  0.64
2013 Adwan L, Zawia NH. Epigenetics: a novel therapeutic approach for the treatment of Alzheimer's disease. Pharmacology & Therapeutics. 139: 41-50. PMID 23562602 DOI: 10.1016/j.pharmthera.2013.03.010  0.64
2012 Alashwal H, Dosunmu R, Zawia NH. Integration of genome-wide expression and methylation data: relevance to aging and Alzheimer's disease. Neurotoxicology. 33: 1450-3. PMID 22743688 DOI: 10.1016/j.neuro.2012.06.008  0.64
2012 Dosunmu R, Alashwal H, Zawia NH. Genome-wide expression and methylation profiling in the aged rodent brain due to early-life Pb exposure and its relevance to aging. Mechanisms of Ageing and Development. 133: 435-43. PMID 22613225 DOI: 10.1016/j.mad.2012.05.003  0.64
2012 Zawia NH, Lahiri DK. Epigenetics: a paradigm shift in understanding Alzheimer's disease. Current Alzheimer Research. 9: 525-6. PMID 22364524  0.64
2012 Maloney B, Sambamurti K, Zawia N, Lahiri DK. Applying epigenetics to Alzheimer's disease via the latent early-life associated regulation (LEARn) model. Current Alzheimer Research. 9: 589-99. PMID 22300406  0.64
2012 Bihaqi SW, Schumacher A, Maloney B, Lahiri DK, Zawia NH. Do epigenetic pathways initiate late onset Alzheimer disease (LOAD): towards a new paradigm. Current Alzheimer Research. 9: 574-88. PMID 22300405  0.64
2012 Bihaqi SW, Zawia NH. Alzheimer's disease biomarkers and epigenetic intermediates following exposure to Pb in vitro. Current Alzheimer Research. 9: 555-62. PMID 22272629  0.64
2011 Bihaqi SW, Huang H, Wu J, Zawia NH. Infant exposure to lead (Pb) and epigenetic modifications in the aging primate brain: implications for Alzheimer's disease. Journal of Alzheimer's Disease : Jad. 27: 819-33. PMID 21891863 DOI: 10.3233/JAD-2011-111013  0.64
2011 Subaiea GM, Alansi BH, Serra DA, Alwan M, Zawia NH. The ability of tolfenamic acid to penetrate the brain: a model for testing the brain disposition of candidate Alzheimer's drugs using multiple platforms. Current Alzheimer Research. 8: 860-7. PMID 21605061 DOI: 10.2174/156720511798192691  0.64
2011 Adwan LI, Basha R, Abdelrahim M, Subaiea GM, Zawia NH. Tolfenamic acid interrupts the de novo synthesis of the β-amyloid precursor protein and lowers amyloid beta via a transcriptional pathway. Current Alzheimer Research. 8: 385-92. PMID 21557719 DOI: 10.2174/156720511795745285  0.64
2011 Huang H, Bihaqi SW, Cui L, Zawia NH. In vitro Pb exposure disturbs the balance between Aβ production and elimination: the role of AβPP and neprilysin. Neurotoxicology. 32: 300-6. PMID 21315759 DOI: 10.1016/j.neuro.2011.02.001  0.64
2010 Zawia NH, Basha R. Developmental Lead Exposure, Epigentics and Late Onset Alzheimer's Disease Developmental Neurotoxicology Research: Principles, Models, Techniques, Strategies, and Mechanisms. 143-162. DOI: 10.1002/9780470917060.ch9  0.64
2009 Lahiri DK, Maloney B, Zawia NH. The LEARn model: An epigenetic explanation for idiopathic neurobiological diseases Molecular Psychiatry. 14: 992-1003. PMID 19851280 DOI: 10.1038/mp.2009.82  0.64
2009 Dosunmu R, Wu J, Adwan L, Maloney B, Basha MR, McPherson CA, Harry GJ, Rice DC, Zawia NH, Lahiri DK. Lifespan profiles of Alzheimer's disease-associated genes and products in monkeys and mice. Journal of Alzheimer's Disease : Jad. 18: 211-30. PMID 19584442 DOI: 10.3233/JAD-2009-1138  0.64
2009 Zawia NH, Lahiri DK, Cardozo-Pelaez F. Epigenetics, oxidative stress, and Alzheimer disease. Free Radical Biology & Medicine. 46: 1241-9. PMID 19245828 DOI: 10.1016/j.freeradbiomed.2009.02.006  0.64
2009 Lahiri DK, Maloney B, Zawia NH. How biochemical pathways for disease may be triggered by early-life events Current Hypotheses and Research Milestones in Alzheimer's Disease. 205-214. DOI: 10.1007/978-0-387-87995-6_17  0.64
2008 Lahiri DK, Zawia NH, Greig NH, Sambamurti K, Maloney B. Early-life events may trigger biochemical pathways for Alzheimer's disease: the "LEARn" model. Biogerontology. 9: 375-9. PMID 18668339 DOI: 10.1007/s10522-008-9162-6  0.64
2008 Royland JE, Wu J, Zawia NH, Kodavanti PR. Gene expression profiles in the cerebellum and hippocampus following exposure to a neurotoxicant, Aroclor 1254: developmental effects. Toxicology and Applied Pharmacology. 231: 165-78. PMID 18602129 DOI: 10.1016/j.taap.2008.04.022  0.64
2008 Brock B, Basha R, DiPalma K, Anderson A, Harry GJ, Rice DC, Maloney B, Lahiri DK, Zawia NH. Co-localization and distribution of cerebral APP and SP1 and its relationship to amyloidogenesis. Journal of Alzheimer's Disease : Jad. 13: 71-80. PMID 18334759  0.64
2008 Abrams DJ, Zheng L, Choo KS, Yang JJ, Wei W, Anchordoquy TJ, Zawia NH, Stevens KE. An initial animal proof-of-concept study for central administration of clozapine to schizophrenia patients. Schizophrenia Research. 100: 86-96. PMID 18178385 DOI: 10.1016/j.schres.2007.10.012  0.64
2008 Wu J, Basha MR, Brock B, Cox DP, Cardozo-Pelaez F, McPherson CA, Harry J, Rice DC, Maloney B, Chen D, Lahiri DK, Zawia NH. Alzheimer's disease (AD)-like pathology in aged monkeys after infantile exposure to environmental metal lead (Pb): evidence for a developmental origin and environmental link for AD. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 28: 3-9. PMID 18171917 DOI: 10.1523/JNEUROSCI.4405-07.2008  0.64
2008 Wu J, Basha MR, Zawia NH. The environment, epigenetics and amyloidogenesis. Journal of Molecular Neuroscience : Mn. 34: 1-7. PMID 18157652 DOI: 10.1007/s12031-007-0009-4  0.64
2007 White LD, Cory-Slechta DA, Gilbert ME, Tiffany-Castiglioni E, Zawia NH, Virgolini M, Rossi-George A, Lasley SM, Qian YC, Basha MR. New and evolving concepts in the neurotoxicology of lead. Toxicology and Applied Pharmacology. 225: 1-27. PMID 17904601 DOI: 10.1016/j.taap.2007.08.001  0.64
2007 Dosunmu R, Wu J, Basha MR, Zawia NH. Environmental and dietary risk factors in Alzheimer's disease. Expert Review of Neurotherapeutics. 7: 887-900. PMID 17610395 DOI: 10.1586/14737175.7.7.887  0.64
2007 Lahiri DK, Maloney B, Basha MR, Ge YW, Zawia NH. How and when environmental agents and dietary factors affect the course of Alzheimer's disease: the "LEARn" model (latent early-life associated regulation) may explain the triggering of AD. Current Alzheimer Research. 4: 219-28. PMID 17430250 DOI: 10.2174/156720507780362164  0.64
2007 Bakheet SA, Basha MR, Cai H, Zawia NH. Lead exposure: expression and activity levels of Oct-2 in the developing rat brain. Toxicological Sciences : An Official Journal of the Society of Toxicology. 95: 436-42. PMID 17093203 DOI: 10.1093/toxsci/kfl163  0.64
2006 Lashuel HA, Zawia N. Islam: governments need to reform education and build a scientific culture. Nature. 444: 545. PMID 17136071 DOI: 10.1038/444545a  0.64
2006 Bolin CM, Basha R, Cox D, Zawia NH, Maloney B, Lahiri DK, Cardozo-Pelaez F. Exposure to lead and the developmental origin of oxidative DNA damage in the aging brain. Faseb Journal : Official Publication of the Federation of American Societies For Experimental Biology. 20: 788-90. PMID 16484331 DOI: 10.1096/fj.05-5091fje  0.64
2006 Donohue M, Wei W, Wu J, Zawia NH, Hud N, De Jesus V, Schmechel D, Hettick JM, Beezhold DH, Vesper S. Characterization of nigerlysin, hemolysin produced by Aspergillus niger, and effect on mouse neuronal cells in vitro. Toxicology. 219: 150-5. PMID 16338047 DOI: 10.1016/j.tox.2005.11.013  0.64
2006 Basha MR, Braddy NS, Zawia NH, Kodavanti PR. Ontogenetic alterations in prototypical transcription factors in the rat cerebellum and hippocampus following perinatal exposure to a commercial PCB mixture. Neurotoxicology. 27: 118-24. PMID 16159668 DOI: 10.1016/j.neuro.2005.07.006  0.64
2005 Zawia NH, Basha MR. Environmental risk factors and the developmental basis for Alzheimer's disease. Reviews in the Neurosciences. 16: 325-37. PMID 16519009  0.64
2005 Basha MR, Murali M, Siddiqi HK, Ghosal K, Siddiqi OK, Lashuel HA, Ge YW, Lahiri DK, Zawia NH. Lead (Pb) exposure and its effect on APP proteolysis and Abeta aggregation. Faseb Journal : Official Publication of the Federation of American Societies For Experimental Biology. 19: 2083-4. PMID 16230335 DOI: 10.1096/fj.05-4375fje  0.64
2005 Basha MR, Wei W, Bakheet SA, Benitez N, Siddiqi HK, Ge YW, Lahiri DK, Zawia NH. The fetal basis of amyloidogenesis: exposure to lead and latent overexpression of amyloid precursor protein and beta-amyloid in the aging brain. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 25: 823-9. PMID 15673661 DOI: 10.1523/JNEUROSCI.4335-04.2005  0.64
2004 Zawia NH, Basha MR, Razmiafshari M. Determining the ability of xenobiotic metals to bind a specific protein domain by electrophoresis Current Protocols in Toxicology / Editorial Board, Mahin D. Maines (Editor-in-Chief) ... [Et Al.]. PMID 23045098  0.64
2003 Zawia NH. Transcriptional involvement in neurotoxicity. Toxicology and Applied Pharmacology. 190: 177-88. PMID 12878047 DOI: 10.1016/S0041-008X(03)00161-3  0.64
2003 Atkins DS, Basha MR, Zawia NH. Intracellular signaling pathways involved in mediating the effects of lead on the transcription factor Sp1 International Journal of Developmental Neuroscience. 21: 235-244. PMID 12850056 DOI: 10.1016/S0736-5748(03)00067-4  0.64
2003 Basha MR, Wei W, Brydie M, Razmiafshari M, Zawia NH. Lead-induced developmental perturbations in hippocampal Sp1 DNA-binding are prevented by zinc supplementation: In vivo evidence for Pb and Zn competition International Journal of Developmental Neuroscience. 21: 1-12. PMID 12565691 DOI: 10.1016/S0736-5748(02)00137-5  0.64
2002 Nayyar T, Zawia NH, Hood DB. Transplacental effects of 2,3,7,8-tetrachlorodibenzo-p-dioxin on the temporal modulation of Sp1 DNA binding in the developing cerebral cortex and cerebellum. Experimental and Toxicologic Pathology : Official Journal of the Gesellschaft FüR Toxikologische Pathologie. 53: 461-8. PMID 11926288 DOI: 10.1078/0940-2993-00219  0.64
2001 Crumpton T, Atkins DS, Zawia NH, Barone S. Lead exposure in pheochromocytoma (PC12) cells alters neural differentiation and Sp1 DNA-binding Neurotoxicology. 22: 49-62. PMID 11307851 DOI: 10.1016/S0161-813X(00)00008-5  0.64
2001 Razmiafshari M, Kao J, D'Avignon A, Zawia NH. NMR identification of heavy metal-binding sites in a synthetic zinc finger peptide: Toxicological implications for the interactions of xenobiotic metals with zinc finger proteins Toxicology and Applied Pharmacology. 172: 1-10. PMID 11264017 DOI: 10.1006/taap.2001.9132  0.64
2000 Zawia NH, Crumpton T, Brydie M, Reddy GR, Razmiafshari M. Disruption of the zinc finger domain: A common target that underlies many of the effects of lead Neurotoxicology. 21: 1069-1080. PMID 11233753  0.64
2000 Reddy GR, Zawia NH. Lead exposure alters Egr-1 DNA-binding in the neonatal rat brain International Journal of Developmental Neuroscience. 18: 791-795. PMID 11154848 DOI: 10.1016/S0736-5748(00)00048-4  0.64
2000 Razmiafshari M, Zawia NH. Utilization of a synthetic peptide as a tool to study the interaction of heavy metals with the zinc finger domain of proteins critical for gene expression in the developing brain Toxicology and Applied Pharmacology. 166: 1-12. PMID 10873713 DOI: 10.1006/taap.2000.8950  0.64
1999 Hilliard A, Ramesh A, Zawia NH. Correlation between lead-induced changes in cerebral ornithine decarboxylase and protein kinase C activities during development and in cultured PC 12 cells International Journal of Developmental Neuroscience. 17: 777-785. PMID 10593613 DOI: 10.1016/S0736-5748(99)00065-9  0.64
1999 Hilliard AL, Ramesh A, Zawia NH. Regional variations in spermine levels in the developing rat brain following exposure to lead Brain Research Bulletin. 49: 349-353. PMID 10452355 DOI: 10.1016/S0361-9230(99)00070-2  0.64
1998 Zawia NH, Sharan R, Brydie M, Oyama T, Crumpton T. Sp1 as a target site for metal-induced perturbations of transcriptional regulation of developmental brain gene expression Developmental Brain Research. 107: 291-298. PMID 9593950 DOI: 10.1016/S0165-3806(98)00023-6  0.64
1997 Sengstock GJ, Zawia NH, Olanow CW, Dunn AJ, Arendash GW. Intranigral iron infusion in the rat - Acute elevations in nigral lipid peroxidation and striatal dopaminergic markers with ensuing nigral degeneration Biological Trace Element Research. 58: 177-195. PMID 9403131 DOI: 10.1007/BF02917470  0.64
1996 Schmitt TJ, Zawia N, Harry GJ. GAP-43 mRNA expression in the developing rat brain: alterations following lead-acetate exposure. Neurotoxicology. 17: 407-14. PMID 8856737  0.64
1996 Harry GJ, Schmitt TJ, Gong Z, Brown H, Zawia N, Evans HL. Lead-induced alterations of glial fibrillary acidic protein (GFAP) in the developing rat brain. Toxicology and Applied Pharmacology. 139: 84-93. PMID 8685912 DOI: 10.1006/taap.1996.0145  0.64
1996 Zawia NH, Harry GJ. Developmental exposure to lead interferes with glial and neuronal differential gene expression in the rat cerebellum. Toxicology and Applied Pharmacology. 138: 43-7. PMID 8658511 DOI: 10.1006/taap.1996.0095  0.64
1995 Zawia NH, Harry GJ. Exposure to lead-acetate modulates the developmental expression of myelin genes in the rat frontal lobe. International Journal of Developmental Neuroscience : the Official Journal of the International Society For Developmental Neuroscience. 13: 639-44. PMID 8553899 DOI: 10.1016/0736-5748(95)00032-C  0.64
1994 Zawia NH, Evers LB, Harry GJ. Developmental profiles of ornithine decarboxylase activity in the hippocampus, neocortex and cerebellum: modulation following lead exposure. International Journal of Developmental Neuroscience : the Official Journal of the International Society For Developmental Neuroscience. 12: 25-30. PMID 8010157 DOI: 10.1016/0736-5748(94)90092-2  0.64
1994 Zawia NH, Evers LB, Kodavanti PR, Harry GJ. Modulation of developmental cerebellar ornithine decarboxylase activity by lead-acetate. Neurotoxicology. 15: 903-11. PMID 7715861  0.64
1993 Zawia NH, Harry GJ. Correlations between developmental ornithine decarboxylase gene expression and enzyme activity in the rat brain. Brain Research. Developmental Brain Research. 71: 53-7. PMID 8431999 DOI: 10.1016/0165-3806(93)90104-I  0.64
1993 Zawia NH, Harry GJ. Trimethyltin-induced c-fos expression: adolescent vs neonatal rat hippocampus. Toxicology and Applied Pharmacology. 121: 99-102. PMID 8337705 DOI: 10.1006/taap.1993.1133  0.64
1992 Zawia NH, Bondy SC. Transcription‐Dependent and ‐Independent Induction of Cerebral Ornithine Decarboxylase Journal of Neurochemistry. 58: 736-739. PMID 1729414 DOI: 10.1111/j.1471-4159.1992.tb09779.x  0.64
1992 Zawia N, Arendash GW, Wecker L. Basal forebrain cholinergic neurons in aged rat brain are more susceptible to ibotenate-induced degeneration than neurons in young adult brain. Brain Research. 589: 333-7. PMID 1393599 DOI: 10.1016/0006-8993(92)91295-P  0.64
1991 Zawia NH, Mattia CJ, Bondy SC. Differential effects of difluoromethylornithine on basal and induced activity of cerebral ornithine decarboxylase and mRNA Neuropharmacology. 30: 337-343. PMID 1852267 DOI: 10.1016/0028-3908(91)90058-J  0.64
1991 Vendrell M, Zawia NH, Serratosa J, Bondy SC. c-fos and ornithine decarboxylase gene expression in brain as early markers of neurotoxicity Brain Research. 544: 291-296. PMID 1710164 DOI: 10.1016/0006-8993(91)90067-6  0.64
1990 Zawia NH, Bondy SC. Electrically stimulated rapid gene expression in the brain: ornithine decarboxylase and c-fos Molecular Brain Research. 7: 243-247. PMID 2159584 DOI: 10.1016/0169-328X(90)90034-B  0.64
1990 Zawia NH, Vendrell M, Bondy SC. Potentiation of stimulated cerebral c-fos and ornithine decarboxylase gene expression by trifluoperazine European Journal of Pharmacology. 183: 1544-1545.  0.64
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