| Year |
Citation |
Score |
| 2023 |
Kotler O, Khrapunsky Y, Shvartsman A, Dai H, Plant LD, Goldstein SAN, Fleidervish I. SUMOylation of Na1.2 channels regulates the velocity of backpropagating action potentials in cortical pyramidal neurons. Elife. 12. PMID 36794908 DOI: 10.7554/eLife.81463 |
0.434 |
|
| 2022 |
Kotler O, Gutnick MJ, Fleidervish IA. In computational models, action potential propagation in ultrathin axons is resilient despite considerable intracellular Na accumulation. Proceedings of the National Academy of Sciences of the United States of America. 119: e2120782119. PMID 35286212 DOI: 10.1073/pnas.2120782119 |
0.584 |
|
| 2021 |
Shvartsman A, Kotler O, Stoler O, Khrapunsky Y, Melamed I, Fleidervish IA. Subcellular distribution of persistent sodium conductance in cortical pyramidal neurons. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. PMID 34099506 DOI: 10.1523/JNEUROSCI.2989-20.2021 |
0.492 |
|
| 2019 |
Revah O, Stoler O, Neef A, Wolf F, Fleidervish IA, Gutnick MJ. Dynamic gain analysis reveals encoding deficiencies in cortical neurons that recover from hypoxia-induced spreading depolarizations. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. PMID 31399533 DOI: 10.1523/Jneurosci.3147-18.2019 |
0.692 |
|
| 2018 |
Katz E, Stoler O, Scheller A, Khrapunsky Y, Goebbels S, Kirchhoff F, Gutnick MJ, Wolf F, Fleidervish IA. Role of sodium channel subtype in action potential generation by neocortical pyramidal neurons. Proceedings of the National Academy of Sciences of the United States of America. PMID 29991598 DOI: 10.1073/Pnas.1720493115 |
0.713 |
|
| 2017 |
Lezmy J, Lipinsky M, Khrapunsky Y, Patrich E, Shalom L, Peretz A, Fleidervish IA, Attali B. M-current inhibition rapidly induces a unique CK2-dependent plasticity of the axon initial segment. Proceedings of the National Academy of Sciences of the United States of America. 114: E10234-E10243. PMID 29109270 DOI: 10.1073/Pnas.1708700114 |
0.51 |
|
| 2016 |
Revah O, Lasser-Katz E, Fleidervish IA, Gutnick MJ. The earliest neuronal responses to hypoxia in the neocortical circuit are glutamate-dependent. Neurobiology of Disease. PMID 27443966 DOI: 10.1016/J.Nbd.2016.07.019 |
0.696 |
|
| 2016 |
Stoler O, Fleidervish IA. Functional implications of axon initial segment cytoskeletal disruption in stroke. Acta Pharmacologica Sinica. 37: 75-81. PMID 26687934 DOI: 10.1038/Aps.2015.107 |
0.41 |
|
| 2015 |
Revah O, Libman L, Fleidervish IA, Gutnick MJ. The Outwardly Rectifying Current of Layer 5 Neocortical Neurons that was Originally Identified as "Non-Specific Cationic" Is Essentially a Potassium Current. Plos One. 10: e0132108. PMID 26197082 DOI: 10.1371/Journal.Pone.0132108 |
0.706 |
|
| 2014 |
Bloch CL, Kedar N, Golan M, Gutnick MJ, Fleidervish IA, Levavi-Sivan B. Long-term GnRH-induced gonadotropin secretion in a novel hypothalamo-pituitary slice culture from tilapia brain. General and Comparative Endocrinology. 207: 21-7. PMID 24859253 DOI: 10.1016/J.Ygcen.2014.05.017 |
0.603 |
|
| 2013 |
Ross W, Fleidervish I, Lasser-Ross N. Imaging sodium in axons and dendrites Cold Spring Harbor Protocols. 8: 433-437. PMID 23637367 DOI: 10.1101/Pdb.Prot074310 |
0.428 |
|
| 2013 |
Baranauskas G, David Y, Fleidervish IA. Spatial mismatch between the Na+ flux and spike initiation in axon initial segment. Proceedings of the National Academy of Sciences of the United States of America. 110: 4051-6. PMID 23341597 DOI: 10.1073/Pnas.1215125110 |
0.459 |
|
| 2013 |
Hofmann D, Neef A, Fleidervish I, Gutnick M, Wolf F. How do channel densities and various time constants affect the dynamic gain of a detailed model of a pyramidal neuron? Bmc Neuroscience. 14. DOI: 10.1186/1471-2202-14-S1-P419 |
0.7 |
|
| 2011 |
Chorin E, Vinograd O, Fleidervish I, Gilad D, Herrmann S, Sekler I, Aizenman E, Hershfinkel M. Upregulation of KCC2 activity by zinc-mediated neurotransmission via the mZnR/GPR39 receptor. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 31: 12916-26. PMID 21900570 DOI: 10.1523/Jneurosci.2205-11.2011 |
0.442 |
|
| 2011 |
Neef A, Wolf F, Gutnick MJ, Fleidervish I. Somatic Sodium Channels Account for Second Phase of Action Potential Upstroke in Soma of Layer 5 pyramidal Cells Biophysical Journal. 100: 15a. DOI: 10.1016/J.Bpj.2010.12.289 |
0.701 |
|
| 2010 |
Fleidervish IA, Lasser-Ross N, Gutnick MJ, Ross WN. Na+ imaging reveals little difference in action potential-evoked Na+ influx between axon and soma. Nature Neuroscience. 13: 852-60. PMID 20543843 DOI: 10.1038/Nn.2574 |
0.684 |
|
| 2008 |
Fleidervish IA, Libman L, Katz E, Gutnick MJ. Endogenous polyamines regulate cortical neuronal excitability by blocking voltage-gated Na+ channels. Proceedings of the National Academy of Sciences of the United States of America. 105: 18994-9. PMID 19020082 DOI: 10.1073/Pnas.0803464105 |
0.711 |
|
| 2008 |
Fleidervish IA, Libman L. How cesium dialysis affects the passive properties of pyramidal neurons: Implications for voltage clamp studies of persistent sodium current New Journal of Physics. 10. DOI: 10.1088/1367-2630/10/3/035001 |
0.548 |
|
| 2006 |
Astman N, Gutnick MJ, Fleidervish IA. Persistent sodium current in layer 5 neocortical neurons is primarily generated in the proximal axon. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 26: 3465-73. PMID 16571753 DOI: 10.1523/Jneurosci.4907-05.2006 |
0.742 |
|
| 2006 |
Binshtok AM, Fleidervish IA, Sprengel R, Gutnick MJ. NMDA receptors in layer 4 spiny stellate cells of the mouse barrel cortex contain the NR2C subunit. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 26: 708-15. PMID 16407568 DOI: 10.1523/Jneurosci.4409-05.2006 |
0.677 |
|
| 2005 |
Levavi-Sivan B, Bloch CL, Gutnick MJ, Fleidervish IA. Electrotonic coupling in the anterior pituitary of a teleost fish. Endocrinology. 146: 1048-52. PMID 15604206 DOI: 10.1210/En.2004-1415 |
0.612 |
|
| 2001 |
Fleidervish IA, Gebhardt C, Astman N, Gutnick MJ, Heinemann U. Enhanced spontaneous transmitter release is the earliest consequence of neocortical hypoxia that can explain the disruption of normal circuit function. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 21: 4600-8. PMID 11425888 DOI: 10.1523/Jneurosci.21-13-04600.2001 |
0.696 |
|
| 2001 |
Fleidervish IA, Gebhardt C, Astman N, Gutnick MJ, Heinemann U. Enhanced Spontaneous Transmitter Release Is the Earliest Consequence of Neocortical Hypoxia That Can Explain the Disruption of Normal Circuit Function The Journal of Neuroscience. 21: 4600-4608. DOI: 10.1523/JNEUROSCI.21-13-04600.2001 |
0.564 |
|
| 1999 |
Levite M, Fleidervish IA, Schwarz A, Pelled D, Futerman AH. Autoantibodies to the glutamate receptor kill neurons via activation of the receptor ion channel Journal of Autoimmunity. 13: 61-72. PMID 10441169 DOI: 10.1006/Jaut.1999.0301 |
0.367 |
|
| 1998 |
Fleidervish IA, Binshtok AM, Gutnick MJ. Functionally distinct NMDA receptors mediate horizontal connectivity within layer 4 of mouse barrel cortex. Neuron. 21: 1055-65. PMID 9856461 DOI: 10.1016/S0896-6273(00)80623-6 |
0.678 |
|
| 1998 |
Astman N, Gutnick MJ, Fleidervish IA. Activation of protein kinase C increases neuronal excitability by regulating persistent Na+ current in mouse neocortical slices. Journal of Neurophysiology. 80: 1547-51. PMID 9744958 DOI: 10.1152/Jn.1998.80.3.1547 |
0.715 |
|
| 1997 |
Kozhinsky E, Fleidervish I, Gutnick M, Golomb D. Transient synchrony in neocortex during propagating activity Neuroscience Letters. 237: S29. DOI: 10.1016/S0304-3940(97)90118-X |
0.595 |
|
| 1997 |
Bibi Y, Fleidervish I, Gutnick M. Early appearance of spontaneous synaptic activity in layer IV neurons of developing mouse barrel cortex Neuroscience Letters. 237: S10. DOI: 10.1016/S0304-3940(97)90041-0 |
0.655 |
|
| 1997 |
Astman N, Fleidervish I, Gutnick M. PKC activation shifts the voltage-dependence of persistent Na+ current in neocortical slices Neuroscience Letters. 237: S4. DOI: 10.1016/S0304-3940(97)90016-1 |
0.669 |
|
| 1996 |
Fleidervish IA, Gutnick MJ. Kinetics of slow inactivation of persistent sodium current in layer V neurons of mouse neocortical slices. Journal of Neurophysiology. 76: 2125-30. PMID 8890326 DOI: 10.1152/Jn.1996.76.3.2125 |
0.718 |
|
| 1996 |
Fleidervish IA, Friedman A, Gutnick MJ. Slow inactivation of Na+ current and slow cumulative spike adaptation in mouse and guinea-pig neocortical neurones in slices. The Journal of Physiology. 493: 83-97. PMID 8735696 DOI: 10.1113/Jphysiol.1996.Sp021366 |
0.71 |
|
| 1995 |
Fleidervish IA, Gutnick MJ. Paired-pulse facilitation of IPSCs in slices of immature and mature mouse somatosensory neocortex. Journal of Neurophysiology. 73: 2591-5. PMID 7666166 DOI: 10.1152/Jn.1995.73.6.2591 |
0.618 |
|
| 1992 |
Undrovinas AI, Fleidervish IA, Makielski JC. Inward sodium current at resting potentials in single cardiac myocytes induced by the ischemic metabolite lysophosphatidylcholine. Circulation Research. 71: 1231-41. PMID 1327577 DOI: 10.1161/01.Res.71.5.1231 |
0.489 |
|
| 1991 |
Burnashev NA, Undrovinas AI, Fleidervish IA, Makielski JC, Rosenshtraukh LV. Modulation of cardiac sodium channel gating by lysophosphatidylcholine. Journal of Molecular and Cellular Cardiology. 23: 23-30. PMID 1645412 DOI: 10.1016/0022-2828(91)90020-M |
0.441 |
|
| 1990 |
Undrovinas AI, Burnashev N, Eroshenko D, Fleidervish I, Starmer CF, Makielski JC, Rosenshtraukh LV. Quinidine blocks adenosine 5'-triphosphate-sensitive potassium channels in heart. The American Journal of Physiology. 259: H1609-12. PMID 2240258 DOI: 10.1152/Ajpheart.1990.259.5.H1609 |
0.403 |
|
| 1989 |
Burnashev NA, Undrovinas AI, Fleidervish IA, Rosenshtraukh LV. Ischemic poison lysophosphatidylcholine modifies heart sodium channels gating inducing long-lasting bursts of openings. PflüGers Archiv : European Journal of Physiology. 415: 124-6. PMID 2560162 DOI: 10.1007/Bf00373151 |
0.442 |
|
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