Samuel L. Pfaff

Affiliations: 
Salk Institute for Biological Studies, La Jolla, CA, United States 
 Neurosciences University of California, San Diego, La Jolla, CA 
Area:
Development
Website:
https://www.salk.edu/scientist/samuel-pfaff/
Google:
"Samuel L. Pfaff, PhD"
Bio:

https://www.hhmi.org/scientists/samuel-l-pfaff

Mean distance: 13.75 (cluster 11)
 
SNBCP
Cross-listing: DevTree

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Publications

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Bravo-Hernandez M, Tadokoro T, Navarro MR, et al. (2019) Spinal subpial delivery of AAV9 enables widespread gene silencing and blocks motoneuron degeneration in ALS. Nature Medicine
Bonanomi D, Valenza F, Chivatakarn O, et al. (2019) p190RhoGAP Filters Competing Signals to Resolve Axon Guidance Conflicts. Neuron
Bohaciakova D, Hruska-Plochan M, Tsunemoto R, et al. (2019) A scalable solution for isolating human multipotent clinical-grade neural stem cells from ES precursors. Stem Cell Research & Therapy. 10: 83
Strnadel J, Carromeu C, Bardy C, et al. (2018) Survival of syngeneic and allogeneic iPSC-derived neural precursors after spinal grafting in minipigs. Science Translational Medicine. 10
Mo Z, Zhao X, Liu H, et al. (2018) Aberrant GlyRS-HDAC6 interaction linked to axonal transport deficits in Charcot-Marie-Tooth neuropathy. Nature Communications. 9: 1007
Hayashi M, Hinckley CA, Driscoll SP, et al. (2018) Graded Arrays of Spinal and Supraspinal V2a Interneuron Subtypes Underlie Forelimb and Hindlimb Motor Control. Neuron
Sternfeld MJ, Hinckley CA, Moore NJ, et al. (2017) Speed and segmentation control mechanisms characterized in rhythmically-active circuits created from spinal neurons produced from genetically-tagged embryonic stem cells. Elife. 6
Hilde KL, Levine AJ, Hinckley CA, et al. (2016) Satb2 Is Required for the Development of a Spinal Exteroceptive Microcircuit that Modulates Limb Position. Neuron
Wasson JA, Simon AK, Myrick DA, et al. (2016) Maternally provided LSD1/KDM1A enables the maternal-to-zygotic transition and prevents defects that manifest postnatally. Elife. 5
He W, Bai G, Zhou H, et al. (2016) Corrigendum: CMT2D neuropathy is linked to the neomorphic binding activity of glycyl-tRNA synthetase. Nature
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