Year |
Citation |
Score |
2016 |
Kino Y, Washizu C, Kurosawa M, Yamada M, Doi H, Takumi T, Adachi H, Katsuno M, Sobue G, Hicks GG, Hattori N, Shimogori T, Nukina N. FUS/TLS acts as an aggregation-dependent modifier of polyglutamine disease model mice. Scientific Reports. 6: 35236. PMID 27739513 DOI: 10.1038/Srep35236 |
0.575 |
|
2015 |
Kino Y, Washizu C, Kurosawa M, Yamada M, Miyazaki H, Akagi T, Hashikawa T, Doi H, Takumi T, Hicks GG, Hattori N, Shimogori T, Nukina N. FUS/TLS deficiency causes behavioral and pathological abnormalities distinct from amyotrophic lateral sclerosis. Acta Neuropathologica Communications. 3: 24. PMID 25907258 DOI: 10.1186/S40478-015-0202-6 |
0.534 |
|
2011 |
Kino Y, Washizu C, Aquilanti E, Okuno M, Kurosawa M, Yamada M, Doi H, Nukina N. Intracellular localization and splicing regulation of FUS/TLS are variably affected by amyotrophic lateral sclerosis-linked mutations. Nucleic Acids Research. 39: 2781-98. PMID 21109527 DOI: 10.1093/Nar/Gkq1162 |
0.51 |
|
2010 |
Bauer PO, Goswami A, Wong HK, Okuno M, Kurosawa M, Yamada M, Miyazaki H, Matsumoto G, Kino Y, Nagai Y, Nukina N. Harnessing chaperone-mediated autophagy for the selective degradation of mutant huntingtin protein. Nature Biotechnology. 28: 256-63. PMID 20190739 DOI: 10.1038/Nbt.1608 |
0.555 |
|
2010 |
Yamanaka T, Tosaki A, Miyazaki H, Kurosawa M, Furukawa Y, Yamada M, Nukina N. Mutant huntingtin fragment selectively suppresses Brn-2 POU domain transcription factor to mediate hypothalamic cell dysfunction. Human Molecular Genetics. 19: 2099-112. PMID 20185558 DOI: 10.1093/Hmg/Ddq087 |
0.555 |
|
2009 |
Miyazaki H, Oyama F, Kurosawa M, Yamada M, Nukina N. Correlation between nuclear accumulation and dysregulation of sodium channel β4 subunit in HD transgenic mice Neuroscience Research. 65: S247. DOI: 10.1016/J.Neures.2009.09.1400 |
0.478 |
|
2008 |
Wong HK, Bauer PO, Kurosawa M, Goswami A, Washizu C, Machida Y, Tosaki A, Yamada M, Knöpfel T, Nakamura T, Nukina N. Blocking acid-sensing ion channel 1 alleviates Huntington's disease pathology via an ubiquitin-proteasome system-dependent mechanism. Human Molecular Genetics. 17: 3223-35. PMID 18658163 DOI: 10.1093/Hmg/Ddn218 |
0.527 |
|
2001 |
Nucifora FC, Sasaki M, Peters MF, Huang H, Cooper JK, Yamada M, Takahashi H, Tsuji S, Troncoso J, Dawson VL, Dawson TM, Ross CA. Interference by huntingtin and atrophin-1 with cbp-mediated transcription leading to cellular toxicity. Science (New York, N.Y.). 291: 2423-8. PMID 11264541 DOI: 10.1126/Science.1056784 |
0.354 |
|
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