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Anne C. Hart

Affiliations: 
Harvard Medical School, Boston, MA, United States 
 Neuroscience Brown University, Providence, RI 
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"Anne Hart"
Mean distance: 15.29 (cluster 11)
 		SNBCP
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Parents

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S. Lawrence Zipursky grad student UCLA

Children

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Ivan C. Alcantara research assistant Brown
Lukas Lu Skuja research assistant 2014-2017 Brown
Gal Haspel post-doc 2004-2006 Harvard Medical School

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Publications

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Walsh MB, Janzen E, Wingrove E, et al. (2020) Genetic modifiers ameliorate endocytic and neuromuscular defects in a model of spinal muscular atrophy. Bmc Biology. 18: 127
Schuch KN, Govindarajan LN, Guo Y, et al. (2020) Discriminating between sleep and exercise-induced fatigue using computer vision and behavioral genetics. Journal of Neurogenetics. 1-13
Carskadon MA, Chappell KR, Barker DH, et al. (2019) A pilot prospective study of sleep patterns and DNA methylation-characterized epigenetic aging in young adults. Bmc Research Notes. 12: 583
Carskadon MA, Chappell KR, Barker DH, et al. (2019) 0022 Prospective Assessment of Sleep and Epigenetic Aging: Preliminary Findings Sleep. 42: A9-A9
Huang H, Hayden DJ, Zhu CT, et al. (2018) Gap Junctions and NCA Cation Channels Are Critical for Developmentally-Timed Sleep and Arousal in . Genetics
Baskoylu SN, Yersak J, O'Hern P, et al. (2018) Single copy/knock-in models of ALS SOD1 in C. elegans suggest loss and gain of function have different contributions to cholinergic and glutamatergic neurodegeneration. Plos Genetics. 14: e1007682
Sorkaç A, DiIorio MA, O'Hern PJ, et al. (2018) LIN-12/Notch Regulates GABA Signaling at the Neuromuscular Junction. G3 (Bethesda, Md.)
Bennett HL, Khoruzhik Y, Hayden D, et al. (2018) Normal sleep bouts are not essential for C. elegans survival and FoxO is important for compensatory changes in sleep. Bmc Neuroscience. 19: 10
Huang H, Zhu CT, Skuja LL, et al. (2017) Genome-Wide Screen for Genes Involved in Caenorhabditis elegans Developmentally Timed Sleep. G3 (Bethesda, Md.)
O'Hern PJ, do Carmo G Gonçalves I, Brecht J, et al. (2017) Decreased microRNA levels lead to deleterious increases in neuronal M2 muscarinic receptors in Spinal Muscular Atrophy models. Elife. 6
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