Farah H. Bardai, Ph.D.

Affiliations: 
2012 Molecular and Cell Biology University of Texas at Dallas, Richardson, TX, United States 
 2013- Pathology Brigham & Women's Hospital/Harvard Medical School, Boston, MA, United States 
Area:
Neurodegenerative diseases, HDACs, Parkinson's Disease
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"Farah Bardai"
Mean distance: 18.08 (cluster 6)
 

Parents

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Santosh D'Mello grad student 2012 UT Dallas
 (Class I histone deacetylase proteins and neurodegeneration.)
Mel  B. Feany post-doc 2013- Harvard Medical School - Brigham and Women's Hospital
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Publications

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Sarkar S, Bardai F, Olsen AL, et al. (2021) Oligomerization of Lrrk controls actin severing and α-synuclein neurotoxicity in vivo. Molecular Neurodegeneration. 16: 33
Bardai FH, Ordonez DG, Bailey RM, et al. (2018) Lrrk promotes tau neurotoxicity through dysregulation of actin and mitochondrial dynamics. Plos Biology. 16: e2006265
Bardai FH, Wang L, Mutreja Y, et al. (2017) A conserved cytoskeletal signaling cascade mediates neurotoxicity of FTDP-17 tau mutations in vivo. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience
Frost B, Bardai FH, Feany MB. (2015) Lamin Dysfunction Mediates Neurodegeneration in Tauopathies. Current Biology : Cb
Bardai FH, Verma P, Smith C, et al. (2013) Disassociation of histone deacetylase-3 from normal huntingtin underlies mutant huntingtin neurotoxicity. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 33: 11833-8
Bardai FH, Price V, Zaayman M, et al. (2012) Histone deacetylase-1 (HDAC1) is a molecular switch between neuronal survival and death. The Journal of Biological Chemistry. 287: 35444-53
Dastidar SG, Bardai FH, Ma C, et al. (2012) Isoform-specific toxicity of Mecp2 in postmitotic neurons: suppression of neurotoxicity by FoxG1. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 32: 2846-55
Bardai FH, D'Mello SR. (2011) Selective toxicity by HDAC3 in neurons: regulation by Akt and GSK3beta. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 31: 1746-51
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