Alan C. Rapraeger, PhD, University of California Berkeley 1979
Affiliations: | Pathology and Laboratory Medicine | University of Wisconsin, Madison, Madison, WI |
Area:
Mechanisms by which the syndecan family of cell surface receptors regulate cell growth, adhesion, and migrationWebsite:
http://www.pathology.wisc.edu/faculty/bio.aspx?name=acrapraegerGoogle:
"Alan Rapraeger"Mean distance: 15.61 (cluster 28) | S | N | B | C | P |
Children
Sign in to add trainee| Charles Yeaman | grad student | 1986-1993 | UW Madison (Cell Biology Tree) |
| Kyle J. McQuade | grad student | 2003 | UW Madison |
| Brandon J. Burbach | grad student | 2004 | UW Madison |
| Benjamin L Allen | grad student | 1998-2004 | UW Madison |
| DeannaLee M. Beauvais | grad student | 2005 | UW Madison |
| Yan Ji | grad student | 2008 | UW Madison |
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Publications
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| Stueven NA, Beauvais DM, Hu R, et al. (2023) Inhibiting IGF1R-mediated Survival Signaling in Head and Neck Cancer with the Peptidomimetic SSTN. Cancer Research Communications. 3: 97-108 |
| Beauvais DM, Nelson SE, Adams KM, et al. (2022) Plasma membrane proteoglycans syndecan-2 and syndecan-4 engage with EGFR and RON kinase to sustain carcinoma cell cycle progression. The Journal of Biological Chemistry. 102029 |
| Jung O, Beauvais DM, Adams KM, et al. (2019) VLA-4 phosphorylation during tumor and immune cell migration relies on its coupling to VEGFR2 and CXCR4 by syndecan-1. Journal of Cell Science |
| Chen M, Choi S, Jung O, et al. (2019) The Specificity of EGF-Stimulated IQGAP1 Scaffold Towards the PI3K-Akt Pathway is Defined by the IQ3 motif. Scientific Reports. 9: 9126 |
| Jung O, Rapraeger A. (2017) Abstract 894: Heparanase-induced shedding of syndecan-1 promotes cancer cell invasion : prevention by inhibitory synstatin peptide Cancer Research. 77: 894-894 |
| Thapa N, Tan X, Choi S, et al. (2016) The Hidden Conundrum of Phosphoinositide Signaling in Cancer. Trends in Cancer. 2: 378-390 |
| Sanderson RD, Elkin M, Rapraeger AC, et al. (2016) Heparanase regulation of cancer, autophagy and inflammation: New mechanisms and targets for therapy. The Febs Journal |
| Beauvais DM, Jung O, Yang Y, et al. (2016) Syndecan-1 (CD138) suppresses apoptosis in multiple myeloma by activating IGF1 receptor: prevention by synstatinIGF1R inhibits tumor growth. Cancer Research |
| Jung O, Trapp-Stamborski V, Purushothaman A, et al. (2016) Heparanase-induced shedding of syndecan-1/CD138 in myeloma and endothelial cells activates VEGFR2 and an invasive phenotype: prevention by novel synstatins. Oncogenesis. 5: e202 |
| Tan X, Lambert PF, Rapraeger AC, et al. (2016) Stress-Induced EGFR Trafficking: Mechanisms, Functions, and Therapeutic Implications. Trends in Cell Biology |