Miguel A. Pappolla, MD PhD
Affiliations: | 2008- | Neurology | University of Texas Medical Branch, Galveston, TX |
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"Miguel Pappolla"Bio:
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Mean distance: 16.34 (cluster 28) | S | N | B | C | P |
Cross-listing: Neuropathology Tree - Alzheimer's Tree
Parents
Sign in to add mentor| Samuel M. Chou | grad student | Cleveland Clinic Foundation | |
| Nikolaos K. Robakis | grad student | ||
| Uros Roessmann | grad student | ||
| Pierluigi C. Gambetti | post-doc | Case Western | |
| George Perry | post-doc | 1985-1986 | Case Western |
Collaborators
Sign in to add collaborator| Blas Frangione | collaborator | ||
| Kumar Sambamurti | collaborator | ||
| Mark Anthony Smith | collaborator | Case Western |
BETA: Related publications
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Publications
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| Pappolla MA, Perry G, Fang X, et al. (2021) Indoles as essential mediators in the gut-brain axis. Their role in Alzheimer's disease. Neurobiology of Disease. 105403 |
| Pappolla MA, Matsubara E, Vidal R, et al. (2018) Melatonin treatment enhances Aβ lymphatic clearance in a transgenic mouse model of amyloidosis. Current Alzheimer Research |
| Baranello RJ, Bharani KL, Padmaraju V, et al. (2015) Amyloid-beta protein clearance and degradation (ABCD) pathways and their role in Alzheimer's disease. Current Alzheimer Research. 12: 32-46 |
| Baranello RJ, Bharani KL, Padmaraju V, et al. (2015) Amyloid-beta protein clearance and degradation (ABCD) pathways and their role in Alzheimer’s disease Current Alzheimer Research. 12: 32-46 |
| Pappolla M, Sambamurti K, Vidal R, et al. (2014) Evidence for lymphatic Aβ clearance in Alzheimer's transgenic mice. Neurobiology of Disease. 71: 215-9 |
| Barnwell E, Padmaraju V, Baranello R, et al. (2014) Evidence of a novel mechanism for partial γ-secretase inhibition induced paradoxical increase in secreted amyloid β protein. Plos One. 9: e91531 |
| Pappolla M, Sambamurti K, Vidal R, et al. (2014) Evidence for lymphatic Aβ clearance in Alzheimer's transgenic mice Neurobiology of Disease. 71: 215-219 |
| Pinnix I, Ghiso JA, Pappolla MA, et al. (2013) Major carboxyl terminal fragments generated by γ-secretase processing of the Alzheimer amyloid precursor are 50 and 51 amino acids long. The American Journal of Geriatric Psychiatry : Official Journal of the American Association For Geriatric Psychiatry. 21: 474-83 |
| Sambamurti K, Greig NH, Utsuki T, et al. (2011) Targets for AD treatment: conflicting messages from γ-secretase inhibitors. Journal of Neurochemistry. 117: 359-74 |
| Poeggeler B, Sambamurti K, Siedlak SL, et al. (2010) A novel endogenous indole protects rodent mitochondria and extends rotifer lifespan. Plos One. 5: e10206 |