Indrapal N. Singh

Affiliations: 
2002-2006 Anatomy & Neurobiology University of Kentucky College of Medicine, Lexington, KY, United States 
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"Indrapal Singh"
Mean distance: 19.8 (cluster 32)
 
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Publications

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Zimmerman AW, Singh K, Connors SL, et al. (2021) Correction to: Randomized controlled trial of sulforaphane and metabolite discovery in children with Autism Spectrum Disorder. Molecular Autism. 12: 44
Zimmerman AW, Singh K, Connors SL, et al. (2021) Randomized controlled trial of sulforaphane and metabolite discovery in children with Autism Spectrum Disorder. Molecular Autism. 12: 38
Hill RL, Singh IN, Wang JA, et al. (2020) Protective effects of phenelzine administration on synaptic and non-synaptic cortical mitochondrial function and lipid peroxidation-mediated oxidative damage following TBI in young adult male rats. Experimental Neurology. 113322
Hill RL, Singh IN, Brelsfoard J, et al. (2020) Pharmacological inhibition of lipid peroxidative damage by the 21-aminosteroid U-74389G improves cortical mitochondrial function following traumatic brain injury in young adult male rats. Neuropharmacology. 108023
Hill RL, Singh IN, Wang J, et al. (2018) THE EFFECTS OF PHENELZINE ADMINISTRATION OF MITOCHONDRIAL FUNCTION, CALCIUM HANDLING AND CYTOSKELETAL DEGRADATION FOLLOWING EXPERIMENTAL TRAUMATIC BRAIN INJURY. Journal of Neurotrauma
Hill RL, Kulbe JR, Singh IN, et al. (2018) Synaptic mitochondria are more susceptible to traumatic brain injury-induced oxidative damage and respiratory dysfunction than non-synaptic mitochondria. Neuroscience
Kulbe JR, Singh IN, Wang J, et al. (2018) CONTINUOUS INFUSION OF PHENELZINE, CYCLOSPORINE A OR THE COMBINATION: EVALUATION OF MITOCHONDRIAL BIOENERGETICS, OXIDATIVE DAMAGE AND CYTOSKELETAL DEGRADATION FOLLOWING SEVERE CONTROLLED CORTICAL IMPACT TRAUMATIC BRAIN INJURY IN RATS. Journal of Neurotrauma
Hill RL, Singh IN, Wang JA, et al. (2017) Time courses of post-injury mitochondrial oxidative damage and respiratory dysfunction and neuronal cytoskeletal degradation in a rat model of focal traumatic brain injury. Neurochemistry International
Cebak JE, Singh IN, Hill RL, et al. (2016) PHENELZINE PROTECTS BRAIN MITOCHONDRIAL FUNCTION In vitro and In vivo FOLLOWING TRAUMATIC BRAIN INJURY BY SCAVENGING THE REACTIVE CARBONYLS 4-HYDROXYNONENAL AND ACROLEIN LEADING TO CORTICAL HISTOLOGICAL NEUROPROTECTION. Journal of Neurotrauma
Kulbe JR, Hill RL, Singh IN, et al. (2016) SYNAPTIC MITOCHONDRIA SUSTAIN MORE DAMAGE THAN NON-SYNAPTIC MITOCHONDRIA FOLLOWING TRAUMATIC BRAIN INJURY AND ARE PROTECTED BY CYCLOSPORINE A. Journal of Neurotrauma
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