Scott R. Whittemore
Affiliations: | Neurological Surgery | University of Louisville, Louisville, KY, United States |
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Publications
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| Forston MD, Wei G, Chariker JH, et al. (2023) Enhanced oxidative phosphorylation, re-organized intracellular signaling, and epigenetic de-silencing as revealed by oligodendrocyte translatome analysis after contusive spinal cord injury. Research Square |
| Gao Y, Wei GZ, Forston MD, et al. (2023) Opposite modulation of functional recovery following contusive spinal cord injury in mice with oligodendrocyte-selective deletions of Atf4 and Chop/Ddit3. Scientific Reports. 13: 9193 |
| Ugiliweneza B, Boakye M, Saraswat Ohri S, et al. (2023) Associations between diurnal timing of spinal cord injury and its etiology and co-morbidities. Journal of Neurotrauma |
| Saraswat Ohri S, Andres K, Howard RM, et al. (2022) Acute pharmacological inhibition of PERK signaling after spinal cord injury spares oligodendrocytes and improves locomotor recovery. Journal of Neurotrauma |
| Whittemore SR, Saraswat Ohri S, Forston MD, et al. (2022) The Proteostasis Network: A Global Therapeutic Target for Neuroprotection after Spinal Cord Injury. Cells. 11 |
| Shepard CT, Pocratsky AM, Brown BL, et al. (2021) Silencing long ascending propriospinal neurons after spinal cord injury improves hindlimb stepping in the adult rat. Elife. 10 |
| Slomnicki LP, Wei G, Burke DA, et al. (2021) Limited changes in locomotor recovery and unaffected white matter sparing after spinal cord contusion at different times of day. Plos One. 16: e0249981 |
| Wei GZ, Saraswat Ohri S, Khattar NK, et al. (2021) Hypoxia-inducible factor prolyl hydroxylase domain (PHD) inhibition after contusive spinal cord injury does not improve locomotor recovery. Plos One. 16: e0249591 |
| Saraswat Ohri S, Burke D, Andres K, et al. (2020) Acute neural and proteostasis mRNA levels predict chronic locomotor recovery after contusive spinal cord injury. Journal of Neurotrauma |
| Saraswat Ohri S, Howard RM, Liu Y, et al. (2020) Oligodendrocyte-specific deletion of Xbp1 exacerbates the endoplasmic reticulum stress response and restricts locomotor recovery after thoracic spinal cord injury. Glia |