Year |
Citation |
Score |
2010 |
Zhao H, Sprunger LK, Simasko SM. Expression of transient receptor potential channels and two-pore potassium channels in subtypes of vagal afferent neurons in rat. American Journal of Physiology. Gastrointestinal and Liver Physiology. 298: G212-21. PMID 19959819 DOI: 10.1152/Ajpgi.00396.2009 |
0.395 |
|
2006 |
Bartoo AC, Sprunger LK, Schneider DA. Expression of sodium channel Nav1.6 in cholinergic myenteric neurons of guinea pig proximal colon. Cell and Tissue Research. 325: 203-9. PMID 16555052 DOI: 10.1007/s00441-006-0179-3 |
0.761 |
|
2006 |
Bartoo AC, Sprunger LK, Schneider DA. Expression of the sodium channel Na(v)1.2 in chemically identified myenteric neurons in the guinea pig. Cell and Tissue Research. 324: 25-32. PMID 16372194 DOI: 10.1007/s00441-005-0107-y |
0.761 |
|
2005 |
Bartoo AC, Sprunger LK, Schneider DA. Expression and distribution of TTX-sensitive sodium channel alpha subunits in the enteric nervous system. The Journal of Comparative Neurology. 486: 117-31. PMID 15844213 DOI: 10.1002/Cne.20541 |
0.751 |
|
2004 |
Meisler MH, Plummer NW, Burgess DL, Buchner DA, Sprunger LK. Allelic mutations of the sodium channel SCN8A reveal multiple cellular and physiological functions. Genetica. 122: 37-45. PMID 15619959 DOI: 10.1007/S10709-004-1441-9 |
0.797 |
|
2003 |
Buchner DA, Trudeau M, George AL, Sprunger LK, Meisler MH. High-resolution mapping of the sodium channel modifier Scnm1 on mouse chromosome 3 and identification of a 1.3-kb recombination hot spot. Genomics. 82: 452-9. PMID 13679025 DOI: 10.1016/S0888-7543(03)00152-6 |
0.738 |
|
2002 |
Kearney JA, Buchner DA, De Haan G, Adamska M, Levin SI, Furay AR, Albin RL, Jones JM, Montal M, Stevens MJ, Sprunger LK, Meisler MH. Molecular and pathological effects of a modifier gene on deficiency of the sodium channel Scn8a (Na(v)1.6). Human Molecular Genetics. 11: 2765-75. PMID 12374766 DOI: 10.1093/Hmg/11.22.2765 |
0.783 |
|
2002 |
Meisler MH, Kearney JA, Sprunger LK, MacDonald BT, Buchner DA, Escayg A. Mutations of voltage-gated sodium channels in movement disorders and epilepsy. Novartis Foundation Symposium. 241: 72-81; discussion 82. PMID 11771652 DOI: 10.1002/0470846682.Ch6 |
0.791 |
|
2001 |
Meisler MH, Kearney J, Escayg A, MacDonald BT, Sprunger LK. Sodium channels and neurological disease: insights from Scn8a mutations in the mouse. The Neuroscientist : a Review Journal Bringing Neurobiology, Neurology and Psychiatry. 7: 136-45. PMID 11496924 DOI: 10.1177/107385840100700208 |
0.812 |
|
2001 |
Maurice N, Tkatch T, Meisler M, Sprunger LK, Surmeier DJ. D1/D5 dopamine receptor activation differentially modulates rapidly inactivating and persistent sodium currents in prefrontal cortex pyramidal neurons. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 21: 2268-77. PMID 11264302 DOI: 10.1523/Jneurosci.21-07-02268.2001 |
0.585 |
|
2000 |
Sprunger LK, Meisler MH, Stratakis CA. Recombination between the sodium channel SCN8A and the Allgrove syndrome gene in a Puerto Rican kindred Journal of Endocrine Genetics. 1: 165-169. |
0.566 |
|
1999 |
Chen K, Sprunger LK, Meisler MH, Waller HJ, Godfrey DA. Reduced spontaneous activity in the dorsal cochlear nucleus of Scn8a mutant mice. Brain Research. 847: 85-9. PMID 10564739 DOI: 10.1016/S0006-8993(99)02031-4 |
0.624 |
|
1999 |
Sprunger LK, Escayg A, Tallaksen-Greene S, Albin RL, Meisler MH. Dystonia associated with mutation of the neuronal sodium channel Scn8a and identification of the modifier locus Scnm1 on mouse chromosome 3. Human Molecular Genetics. 8: 471-9. PMID 9949206 DOI: 10.1093/Hmg/8.3.471 |
0.754 |
|
1998 |
Plummer NW, Galt J, Jones JM, Burgess DL, Sprunger LK, Kohrman DC, Meisler MH. Exon organization, coding sequence, physical mapping, and polymorphic intragenic markers for the human neuronal sodium channel gene SCN8A. Genomics. 54: 287-96. PMID 9828131 DOI: 10.1006/Geno.1998.5550 |
0.754 |
|
1998 |
GarcÃa KD, Sprunger LK, Meisler MH, Beam KG. The sodium channel Scn8a is the major contributor to the postnatal developmental increase of sodium current density in spinal motoneurons. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 18: 5234-9. PMID 9651206 DOI: 10.1523/Jneurosci.18-14-05234.1998 |
0.563 |
|
1998 |
Meisler MH, Griffith AJ, Warman M, Tiller G, Sprunger LK. Gene symbol: COL11A1. Disease: Marshall syndrome. Human Genetics. 102: 498. PMID 9600252 DOI: 10.1007/S004390050731 |
0.476 |
|
1998 |
Griffith AJ, Sprunger LK, Sirko-Osadsa DA, Tiller GE, Meisler MH, Warman ML. Marshall syndrome associated with a splicing defect at the COL11A1 locus. American Journal of Human Genetics. 62: 816-23. PMID 9529347 DOI: 10.1086/301789 |
0.519 |
|
1997 |
Meisler MH, Sprunger LK, Plummer NW, Escayg A, Jones JM. Ion channel mutations in mouse models of inherited neurological disease. Annals of Medicine. 29: 569-74. PMID 9562526 DOI: 10.3109/07853899709007484 |
0.794 |
|
1997 |
Raman IM, Sprunger LK, Meisler MH, Bean BP. Altered subthreshold sodium currents and disrupted firing patterns in Purkinje neurons of Scn8a mutant mice. Neuron. 19: 881-91. PMID 9354334 DOI: 10.1016/S0896-6273(00)80969-1 |
0.598 |
|
1996 |
Sprunger LK, Stewig NJ, O'Grady SM. Effects of charybdotoxin on K+ channel (KV1.2) deactivation and inactivation kinetics. European Journal of Pharmacology. 314: 357-64. PMID 8957259 DOI: 10.1016/S0014-2999(96)00556-0 |
0.638 |
|
1996 |
Sprunger LK, O'Grady SM. Properties of a delayed-rectifier K+ channel in a mouse nonfusing muscle cell line Cellular Physiology and Biochemistry. 6: 210-222. |
0.637 |
|
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