Eliezer Masliah, MD, National Autonomous University of Mexico 1983

Affiliations: 
Pathology and Medicine University of California, San Diego, La Jolla, CA 
Area:
http://www.researchprofiles.collexis.com/jad/expert.asp?u_id=7
Website:
http://molpath.ucsd.edu/faculty/Masliah.shtml
Google:
"Eliezer Masliah"
Bio:

http://www.researchprofiles.collexis.com/jad/expert.asp?u_id=7

Mean distance: 16.1 (cluster 32)
 
SNBCP
Cross-listing: Neuropathology Tree - Alzheimer's Tree

BETA: Related publications

Publications

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Hashimoto M, Ho G, Takamatsu Y, et al. (2020) Understanding Creutzfeldt-Jackob disease from a viewpoint of amyloidogenic evolvability. Prion. 14: 1-8
Iba M, Kim C, Florio J, et al. (2020) Role of Alterations in Protein Kinase p38γ in the Pathogenesis of the Synaptic Pathology in Dementia With Lewy Bodies and α-Synuclein Transgenic Models. Frontiers in Neuroscience. 14: 286
Waragai M, Ho G, Takamatsu Y, et al. (2020) Adiponectin Paradox in Alzheimer's Disease; Relevance to Amyloidogenic Evolvability? Frontiers in Endocrinology. 11: 108
Marschallinger J, Altendorfer B, Rockenstein E, et al. (2020) The Leukotriene Receptor Antagonist Montelukast Reduces Alpha-Synuclein Load and Restores Memory in an Animal Model of Dementia with Lewy Bodies. Neurotherapeutics : the Journal of the American Society For Experimental Neurotherapeutics
Hashimoto M, Ho G, Takamatsu Y, et al. (2019) Possible Role of Amyloid Cross-Seeding in Evolvability and Neurodegenerative Disease. Journal of Parkinson's Disease
Schofield DJ, Irving L, Calo L, et al. (2019) Preclinical development of a high affinity α-synuclein antibody, MEDI1341, that can enter the brain, sequester extracellular α-synuclein and attenuate α-synuclein spreading in vivo. Neurobiology of Disease. 104582
Kunkle BW, Grenier-Boley B, Sims R, et al. (2019) Author Correction: Genetic meta-analysis of diagnosed Alzheimer's disease identifies new risk loci and implicates Aβ, tau, immunity and lipid processing. Nature Genetics
Mandler M, Rockenstein E, Overk C, et al. (2019) Effects of single and combined immunotherapy approach targeting amyloid β protein and α-synuclein in a dementia with Lewy bodies-like model. Alzheimer's & Dementia : the Journal of the Alzheimer's Association
He X, Yang W, Zeng Z, et al. (2019) NLRP3-dependent pyroptosis is required for HIV-1 gp120-induced neuropathology. Cellular & Molecular Immunology
Gerez JA, Prymaczok NC, Rockenstein E, et al. (2019) A cullin-RING ubiquitin ligase targets exogenous α-synuclein and inhibits Lewy body-like pathology. Science Translational Medicine. 11
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