Aaron DiAntonio
Affiliations: | Washington University, Saint Louis, St. Louis, MO |
Area:
Drosophila NMJGoogle:
"Aaron DiAntonio"Mean distance: 13.54 (cluster 32) | S | N | B | C | P |
Parents
Sign in to add mentorThomas L. Schwarz | grad student | Stanford Medical School | |
Corey S. Goodman | post-doc | UC Berkeley |
Children
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Publications
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Lee B, Oh Y, Cho E, et al. (2022) FK506-binding protein-like and FK506-binding protein 8 regulate dual leucine zipper kinase degradation and neuronal responses to axon injury. The Journal of Biological Chemistry. 101647 |
Bloom AJ, Mao X, Strickland A, et al. (2022) Constitutively active SARM1 variants that induce neuropathy are enriched in ALS patients. Molecular Neurodegeneration. 17: 1 |
Li Y, Pazyra-Murphy MF, Avizonis D, et al. (2022) Sarm1 activation produces cADPR to increase intra-axonal Ca++ and promote axon degeneration in PIPN. The Journal of Cell Biology. 221 |
Ko KW, Devault L, Sasaki Y, et al. (2021) Live imaging reveals the cellular events downstream of SARM1 activation. Elife. 10 |
Wu T, Zhu J, Strickland A, et al. (2021) Neurotoxins subvert the allosteric activation mechanism of SARM1 to induce neuronal loss. Cell Reports. 37: 109872 |
Eastman S, Smith T, Zaydman MA, et al. (2021) A phytobacterial TIR domain effector manipulates NAD to promote virulence. The New Phytologist |
DiAntonio A, Milbrandt J, Figley MD. (2021) The SARM1 TIR NADase: Mechanistic Similarities to Bacterial Phage Defense and Toxin-Antitoxin Systems. Frontiers in Immunology. 12: 752898 |
Sasaki Y, Zhu J, Shi Y, et al. (2021) Nicotinic acid mononucleotide is an allosteric SARM1 inhibitor promoting axonal protection. Experimental Neurology. 113842 |
Figley MD, Gu W, Nanson JD, et al. (2021) SARM1 is a metabolic sensor activated by an increased NMN/NAD ratio to trigger axon degeneration. Neuron |
Chen YH, Sasaki Y, DiAntonio A, et al. (2021) SARM1 is required in human derived sensory neurons for injury-induced and neurotoxic axon degeneration. Experimental Neurology. 113636 |