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Christopher D Nelson, PhD

Affiliations: 
2001-2007 Biochemistry Duke University, Durham, NC 
 2008-2009 Neuroscience Massachusetts Institute of Technology, Cambridge, MA, United States 
 2009-2013 Neuroscience Genentech, Inc., San Francisco, CA, United States 
 2014- Diagnostics Roche, Indianapolis, IN, United States 
Area:
GPCRs, arrestin, neuroscience, diagnostics
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Parents

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Robert J. Lefkowitz grad student 2002-2007 Duke
Morgan Sheng post-doc 2008-2013 MIT / Genentech

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Publications

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Nelson CD, Sheng M. (2013) Gpr3 stimulates Aβ production via interactions with APP and β-arrestin2. Plos One. 8: e74680
Nelson CD, Kim MJ, Hsin H, et al. (2013) Phosphorylation of threonine-19 of PSD-95 by GSK-3β is required for PSD-95 mobilization and long-term depression. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 33: 12122-35
Boudreau AC, Milovanovic M, Conrad KL, et al. (2012) A protein cross-linking assay for measuring cell surface expression of glutamate receptor subunits in the rodent brain after in vivo treatments. Current Protocols in Neuroscience / Editorial Board, Jacqueline N. Crawley ... [Et Al.]. Unit 5.30.1-19
Chen X, Nelson CD, Li X, et al. (2011) PSD-95 is required to sustain the molecular organization of the postsynaptic density. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 31: 6329-38
Ozawa K, Whalen EJ, Nelson CD, et al. (2008) S-nitrosylation of beta-arrestin regulates beta-adrenergic receptor trafficking. Molecular Cell. 31: 395-405
Nelson CD, Kovacs JJ, Nobles KN, et al. (2008) Beta-arrestin scaffolding of phosphatidylinositol 4-phosphate 5-kinase Ialpha promotes agonist-stimulated sequestration of the beta2-adrenergic receptor. The Journal of Biological Chemistry. 283: 21093-101
Whalen EJ, Foster MW, Matsumoto A, et al. (2007) Regulation of beta-adrenergic receptor signaling by S-nitrosylation of G-protein-coupled receptor kinase 2. Cell. 129: 511-22
Nelson CD, Perry SJ, Regier DS, et al. (2007) Targeting of diacylglycerol degradation to M1 muscarinic receptors by beta-arrestins. Science (New York, N.Y.). 315: 663-6
Gesty-Palmer D, Chen M, Reiter E, et al. (2006) Distinct beta-arrestin- and G protein-dependent pathways for parathyroid hormone receptor-stimulated ERK1/2 activation. The Journal of Biological Chemistry. 281: 10856-64
Shenoy SK, Drake MT, Nelson CD, et al. (2006) beta-arrestin-dependent, G protein-independent ERK1/2 activation by the beta2 adrenergic receptor. The Journal of Biological Chemistry. 281: 1261-73
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