Year |
Citation |
Score |
2011 |
Davis BK, Roberts RA, Huang MT, Willingham SB, Conti BJ, Brickey WJ, Barker BR, Kwan M, Taxman DJ, Accavitti-Loper MA, Duncan JA, Ting JP. Cutting edge: NLRC5-dependent activation of the inflammasome. Journal of Immunology (Baltimore, Md. : 1950). 186: 1333-7. PMID 21191067 DOI: 10.4049/Jimmunol.1003111 |
0.637 |
|
2009 |
Willingham SB, Allen IC, Bergstralh DT, Brickey WJ, Huang MT, Taxman DJ, Duncan JA, Ting JP. NLRP3 (NALP3, Cryopyrin) facilitates in vivo caspase-1 activation, necrosis, and HMGB1 release via inflammasome-dependent and -independent pathways. Journal of Immunology (Baltimore, Md. : 1950). 183: 2008-15. PMID 19587006 DOI: 10.4049/Jimmunol.0900138 |
0.767 |
|
2009 |
Duncan JA, Gao X, Huang MT, O'Connor BP, Thomas CE, Willingham SB, Bergstralh DT, Jarvis GA, Sparling PF, Ting JP. Neisseria gonorrhoeae activates the proteinase cathepsin B to mediate the signaling activities of the NLRP3 and ASC-containing inflammasome. Journal of Immunology (Baltimore, Md. : 1950). 182: 6460-9. PMID 19414800 DOI: 10.4049/Jimmunol.0802696 |
0.772 |
|
2009 |
Huang MT, Taxman DJ, Holley-Guthrie EA, Moore CB, Willingham SB, Madden V, Parsons RK, Featherstone GL, Arnold RR, O'Connor BP, Ting JP. Critical role of apoptotic speck protein containing a caspase recruitment domain (ASC) and NLRP3 in causing necrosis and ASC speck formation induced by Porphyromonas gingivalis in human cells. Journal of Immunology (Baltimore, Md. : 1950). 182: 2395-404. PMID 19201894 DOI: 10.4049/Jimmunol.0800909 |
0.707 |
|
2008 |
Willingham SB, Ting JP. NLRs and the dangers of pollution and aging. Nature Immunology. 9: 831-3. PMID 18645588 DOI: 10.1038/ni0808-831 |
0.346 |
|
2008 |
Li H, Willingham SB, Ting JP, Re F. Cutting edge: inflammasome activation by alum and alum's adjuvant effect are mediated by NLRP3. Journal of Immunology (Baltimore, Md. : 1950). 181: 17-21. PMID 18566365 |
0.681 |
|
2008 |
Ting JP, Willingham SB, Bergstralh DT. NLRs at the intersection of cell death and immunity. Nature Reviews. Immunology. 8: 372-9. PMID 18362948 DOI: 10.1038/Nri2296 |
0.777 |
|
2007 |
Willingham SB, Bergstralh DT, O'Connor W, Morrison AC, Taxman DJ, Duncan JA, Barnoy S, Venkatesan MM, Flavell RA, Deshmukh M, Hoffman HM, Ting JP. Microbial pathogen-induced necrotic cell death mediated by the inflammasome components CIAS1/cryopyrin/NLRP3 and ASC. Cell Host & Microbe. 2: 147-59. PMID 18005730 DOI: 10.1016/J.Chom.2007.07.009 |
0.764 |
|
2007 |
Duncan JA, Bergstralh DT, Wang Y, Willingham SB, Ye Z, Zimmermann AG, Ting JP. Cryopyrin/NALP3 binds ATP/dATP, is an ATPase, and requires ATP binding to mediate inflammatory signaling. Proceedings of the National Academy of Sciences of the United States of America. 104: 8041-6. PMID 17483456 DOI: 10.1073/Pnas.0611496104 |
0.743 |
|
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